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Shannon [1] first proposed the idea of entropy in 1948. It is a fundamental idea in the theories of information that measures the degree of uncertainty or knowledge contained in a given random variable. Shannon entropy is acquainted mathematically as
$ Sn(P)=−n∑k=1pklnpk, $ |
for discrete random variables, with noting that $ \textbf{P} = (p_{1}, p_{2}, ..., p_{n}) $ is the vector of mass function probability, or
$ Sn(X)=−∫∞−∞f(x)lnf(x)dx, $ | (1.1) |
for a randomly generated continuous variable $ X $, and $f(x)$ represents the probability density functional (PDF). It has proven to be a powerful tool in various fields, including communication systems, where it optimizes data transmission, and in data compression, where it measures the limits of compressibility. By capturing the average amount of information generated by random events, Shannon entropy provides a robust framework for analyzing uncertainty in diverse systems.
Building on this seminal work, Rao et al. [2] established the conception of the cumulative residual entropy model as an extension of entropy designed for survival analysis and reliability theory. Unlike traditional entropy, which measures overall uncertainty, residual cumulative entropy focuses on the uncertainty remaining in a system or process beyond a given time or threshold. Formally, given a randomly variable that is not negative $X$, the residual cumulative function of entropy measurement is described as:
$ RCEn(X)=∫∞0ˉF(x)lnˉF(x)dx, $ | (1.2) |
in which the survival function is encapsulated by $\bar{F}(x) = 1 - F(x)$, and the cumulative distribution function (CDF) of $X$ is encapsulated by $F(x)$. This measure captures the tail behavior of the determined distribution, causing it to be quite helpful in applications requiring an understanding of the uncertainty associated with extreme events. Residual cumulative entropy has since been employed in numerous disciplines regarding reliability engineering, risk analysis form, and lifetime data analysis, where the quantification of remaining uncertainty is of critical importance.
Following Campbell [3], Pal and Pal [4,5] used these concerns to establish a new measure, called the exponential entropy measure, through other descriptions parallel to Shannon entropy. In the discrete situation, the formulation of the exponential entropy model is described below:
$ EXn(P)=n∑k=1Pk(e1−Pk−1). $ | (1.3) |
They added the $ -1 $ term since it seems only logical that any measurement that contains data should be assigned $ 0 $ for the degenerative distribution of probabilities $ (0, ..., 0, 1, 0, ..., 0) $. The authors argued that exponential entropy offers distinct benefits over Shannon's formulation. For example, they noted that exponential entropy reaches a fixed upper value in the case of a distribution that is considered to be uniform, where each probability is given by $p_{k} = \frac{1}{n}$ for $k = 1, 2, \dots, n$ as demonstrated by
$ \lim\limits_{n\rightarrow \infty}EXn\left(\frac{1}{n}, \frac{1}{n}, \dots, \frac{1}{n}\right) = e-1; $ |
in contrast, Shannon's entropy does not exhibit this bounded behavior.
Furthermore, Panjehkeh et al. [6] examined the features and attributes of the exponential measure of entropy, along with the Shannon measure of entropy model, in both discrete and continuous situations, including the asymptotic equipartition property, invariancy under monotone transformation, subadditivity, and the chain rule. The concept of a continuous exponential measure of entropy was first presented as
$ EXn(X)=∫Dsf(x)(e1−f(x))dx. $ | (1.4) |
We may observe that Panjehkeh et al. [6] eliminated the term $ -1 $ from the measurement in order to make it inherently non-negative against Shannon entropy, given that $ X $ is a randomly variable that is continuous, following the PDF $ f $, and the support $ D_{s} $. Kvalseth [7] developed a more broad variant of the exponential entropy of Eq (1.2), known as generalized entropy, given by
$ GEXn(P)=1βn∑k=1pk(e1−pβk−1), $ | (1.5) |
where the parameter $ \beta $ is arbitrary and has a non-zero real value (i.e., $ \beta\in \mathbb{R}\backslash\{0\} $). Moreover, the generalized exponential entropy measure has been used in many topics. Alotaibi and Elaraby [8] created a generalized exponential entropy-based method for COVID-19 disease segmentation from computed tomography scans. Fuzzy $ c $ partitioning and generalized exponential entropy were combined in their suggested method. In multi-criteria decision-making, we can see, for example, Wei et al. [9] and Dinesh and Kumar [10].
Work motivation: Since the PDF is calculated as the distribution's derivative form, the CDF appears more regularly than the density function. Furthermore, the CDF is what is relevant and/or quantifiable in practice. For instance, if the variable that is chosen randomly is the life span of a machine, the happenstance that is relevant is not whether the life span equals $ t $, but rather whether it surpasses $ t $. This is what prompted us to study it instead of the existing entropy based on the PDF. The question that arises is whether it can be generalized and if the traditional model can be made a special case of it. Moreover, studying the discrete case is not satisfactory without considering the continuous case. As we have mentioned that the exponential entropy in discrete case has been discussed in distinct fields; take into consider, for instance, Wei et al. [11], and Ye and Cui [12]. Therefore, it is essential to study and discuss it in the continuous case with clarification of the applications associated with the continuous side. Another important aspect to discuss is that the proposed model is based on the exponential function. Dose this function help solve some of the problems that appear in uncertainty issues?
This article aimed to present a generalization of the continuous residual cumulative entropy, known as residual cumulative generalized exponential entropy. Applications, including non-parametric estimation, are provided. On the other hand, the continuous form of the generalized exponential entropy is used to discuss the characterization of symmetry via order statistics. Moreover, a classification problem involving the pattern recognition in diabetes data based on the generalized exponential entropy model is presented. The following is the arrangement of the remaining portions of this paper: in Section 2, the concept of residual cumulative generalized exponential entropy in the continuous setting is presented. Additionally, the properties, including bounds, non-negativity, the relationship with differential entropy, and stochastic orders, are explained. In Section 3, some consequences of the residual cumulative generalized exponential entropy expansion, such as the Bayes risk and the order of excess wealth, are discussed. In Section 4, the non-parametric estimation of the empirical residual cumulative generalized exponential entropy is applied using two methods. Finally, in Section 5, the symmetry characterization using order statistics with an example based on the symmetrical uniform distribution and the classification problem using pattern recognition based on the generalized exponential entropy are illustrated.
In this section, we will establish the concept of the residual cumulative generalized exponential entropy. Inspired by Rao et al. [2], we can depend on the function of survival $ \bar{F}(x) $ to derive the residual cumulative generalized exponential entropy from the discrete case of the generalized exponential entropy in (1.5) according to the following definition.
Definition 2.1. Consider the non-negativity continual randomly variable $ X $ following the CDF $ F $. Then, we can realize the residual cumulative generalized exponential entropy by the following formula:
$ RGEXnβ(X)=1β∫∞0ˉF(x)(e1−ˉFβ(x)−1)dx=1β[∫∞0ˉF(x)e1−ˉFβ(x)dx−μ], $ | (2.1) |
where $ \beta\in \mathbb{R}\backslash\{0\} $, and the mean (expected value) $ \mu = E(X) = \int_{0}^{\infty} \bar{F}(x) \, dx $.
The following proposition shows the limitation of the residual cumulative generalized exponential entropy when $ \beta $ tends to zero, which returns to residual cumulative entropy in (1.2).
Proposition 2.1. Consider the non-negativity continual random variable $ X $ following the CDF $ F $. Then, from (2.1) and (1.2), we have
$ limβ→0RGEXnβ(X)=RCEn(X). $ |
Proof. From (2.1), utilizing the L'Hopital's rule, we have
$ limβ→0RGEXnβ(X)=limβ→0∫∞0ˉF(x)(e1−ˉFβ(x)−1)dxβ=limβ→0−∫∞0ˉF1+β(x)e1−ˉFβ(x)lnˉF(x)dx=∫∞0ˉF(x)lnˉF(x)dx=RCEn(X). $ |
In the following discussions, we will discuss the non-negativity and the bounds of the residual cumulative generalized exponential entropy when $ \beta > 0 $.
Proposition 2.2. Consider the non-negativity continual random variable $ X $ following the CDF $ F $. Then, from (2.1), we can say that
(1) The residual cumulative generalized exponential entropy is non-negative for all $ \beta > 0 $.
(2) To discuss the bounds of the residual cumulative generalized exponential entropy:
(a) We get
$ RGEXnβ(X)≥(≤)RGEXn1(X),∀0<β≤1(β≥1). $ | (2.2) |
(b) We get
$ 0≤RGEXnβ(X)≤μ(e−1)β,∀β≥0. $ | (2.3) |
Proof. $ (1) $ Its known that $ e^{1-\bar{F}^{\beta}(x)}\geq 1 $, for all $ \beta > 0 $, and $ \bar{F}(x)\in [0, 1] $. Thus, $ \bar{F}(x) \, e^{1-\bar{F}^{\beta}(x)}\geq \bar{F}(x) $, which implies that $ \int_{0}^{\infty} \bar{F}(x) \, e^{1-\bar{F}^{\beta}(x)} \, dx\geq \int_{0}^{\infty} \bar{F}(x) \, dx = \mu $. Then, $ RGEXn_{\beta}(X)\geq0 $, for all $ \beta > 0 $. Or, by another method, we can assume, by converse, that $ \beta \, RGEXn_{\beta}(X) < 0 $, then we have
$ ∫∞0ˉF(x)e1−ˉFβ(x)dx−∫∞0ˉF(x)dx<0⇒∫∞0ˉF(x)e1−ˉFβ(x)dx<∫∞0ˉF(x)e0dx. $ |
Therefore, we deduce that $ e^{1-\bar{F}^{\beta}(x)} < 1 $, which implies $ 1 - \bar{F}^{\beta}(x) < 0 $ or, equivalently, $ \bar{F}^{\beta}(x) > 1 $. This contradicts the fact that $\bar{F}(x) \in [0, 1]$ for any $x$. Then, the result follows.
$ (2) $ For $ 0 < \beta\leq1 (\beta\geq1) $, we have $ \bar{F}^{\beta}(x)\geq (\geq) \bar{F}(x) $, which implies that $ \bar{F}(x) \, e^{1-\bar{F}^{\beta}(x)}\geq (\leq) \bar{F}(x) e^{1-\bar{F}(x)} $. Then, $ \frac{1}{\beta} \left[\int_{0}^{\infty} \bar{F}(x) e^{1-\bar{F}^{\beta}(x)} \, dx-\mu\right]\geq (\leq) \int_{0}^{\infty} \bar{F}(x) e^{1-\bar{F}(x)} \, dx-\mu $, and the result follows.
Lemma 2.1. If $ \mu = E(X) < \infty $, then $ RGEXn_{\beta}(X) < \infty $, for all $ \beta > 0 $.
Proof. The result is obtained directly from (2.3).
Theorem 2.1. If $ X $ is an absolutely continual non-negativity random variable following a PDF $ f(x) $, then
$ RGEXn_{1}(X) \geq C^{*} e^{Sn(X)}, $ |
with noting that
$ C^{*} = \exp\left\{ \int_0^1 \ln \left|u\left(e^{1-u}-1\right)\right| du\right\}\simeq 0.176192, $ |
and $ Sn(X) $ is defined in (1.1).
Proof. Using the fact of the inequality of log-sum, it contends that
$ ∫∞0f(x)lnf(x)|¯F(x)(e1−¯F(x)−1)|dx≥ln1∫∞0|¯F(x)(e1−¯F(x)−1)|dxdx=−lnRGEXn(X), $ | (2.4) |
Moreover, the left-hand side of (2.4) can be expressed as
$ ∫∞0f(x)lnf(x)|¯F(x)(e1−¯F(x)−1)|dx=−Sn(X)−∫∞0f(x)ln|¯F(x)(e1−¯F(x)−1)|dx=−Sn(X)−∫10ln|u(e1−u−1)|du. $ |
Therefore, it follows that
$ Sn(X) + \int_0^1 \ln \left|u\left(e^{1-u}-1\right)\right| du \leq \ln RGEXn(X). $ |
Applying the exponential function to both sides of the aforementioned relation, we derive
$ RGEXn(X) \geq C^{*} e^{Sn(X)}, $ |
where
$ C^{*} = \exp\left\{ \int_0^1 \ln \left|u\left(e^{1-u}-1\right)\right| du\right\}\simeq 0.176192, $ |
thereby finalizing the proof.
Proposition 2.3. If $ X $ is an absolutely continual non-negativity random variable with residual cumulative generalized exponential entropy $ RGEXn_{\beta}(X) < \infty $ as given in (2.1), $ \forall \beta > 0 $. Then, we can obtain
$ RGEXnβ(X)=1βE(ψβ(X)), $ | (2.5) |
where
$ ψβ(X)=∫x0(e1−ˉFβ(t)−1)dt. $ | (2.6) |
Proof. From (2.1), and utilizing the theorem of Fubini, we can express the following:
$ RGEXnβ(X)=1β∫∞0[∫∞tf(x)dx](e1−ˉFβ(t)−1)dt=1β∫∞0f(x)[∫x0(e1−ˉFβ(t)−1)dt]dx, $ |
and (2.5) is obtained by utilizing (2.6).
In the following, we will show some examples of the residual cumulative generalized exponential entropy of well-known distributions.
Example 2.1. Consider the non-negativity continual random variable $ X $ following the CDF $ F $. Then,
(1) Under the distribution of exponential ($ Exp(\gamma) $) with $ F(x) = 1-e^{-\gamma x} $, we get
$ RGEXnβ(X)=−(β−eΓ[1β]+eΓ[1β,1])γβ2, $ |
where $ \Gamma\left[\frac{1}{\beta}\right] $ is the function of the Euler gamma, and $ \Gamma\left[\frac{1}{\beta}, 1\right] $ is the function of the incomplete gamma. Moreover, with $ \beta = 1 $, we get
$ RGEXn1(X)=2−eγ. $ |
(2) Under the uniform distribution ($ Ud(t_{1}, t_{2}) $) with $ F(x) = \frac{x-t_{1}}{t_{2}-t_{1}} $, and $ \beta = 1 $, we get
$ RGEXn1(X)=e−52. $ |
Figure 1 shows the plot of the residual cumulative generalized exponential entropy and the residual cumulative entropy defined in (1.2) of the $ Exp(\gamma) $ distribution. Therefore, we can see the assurance of Proposition 2.1 and Eq (2.2).
The theorem outlined below establishes a characterization in terms of the residual cumulative generalized exponential entropy under the usual stochastic, dispersive, increasing convex, and hazard rate orders. In the usual stochastic, dispersive, increasing convex, and hazard rate orders, represented, respectively, by $X_{1} \leq_{\text{UstOr}} X_{2}$, $X_{1} \leq_{\text{DisOr}} X_{2}$, $X_{1} \leq_{\text{IcxOr}} X_{2}$, and $X_{1} \leq_{\text{HrOr}} X_{2}$, we mainly remember that the random variable $X_{1}$ is smaller than $X_{2}$ if
$ (1) \; \bar{F}_{1}(x)\leq \bar{F}_{2}(x) $ (for the usual stochastic order),
$ (2) \; F_{2}^{-1}(v) - F_{1}^{-1}(v) \text{ is increasing in } v \in (0, 1) $ (for the dispersive order),
$ (3) \; E(\xi(X_{1})) = E(\xi(X_{1})) $, with the existence of the expectations and for all convex increasing functions $ \xi $ (for the increasing convex order),
$ (4) \; \frac{\bar{F}_{2}(x)}{\bar{F}_{1}(x)} $ is increasing with respect to $ x $ (for the hazard rate order).
where $F_{1}^{-1}$ and $F_{2}^{-1}$ are the right continually inverses of the CDF's $F_{1}$ and $F_{2}$, correspondingly; see Shaked and Shanthikumar [13].
Theorem 2.2. Assume that two random variables that are continuously, $X_{1}$ and $X_{2}$, following distribution functions that are strictly increasing, ($F_{1}$ and $F_{2}$, respectively). If $X_{1} \leq_{\text{UstOr}} X_{2}$, then, we obtain:
(1) $ RGEXn_{\beta}(X_{1})\leq RGEXn_{\beta}(X_{2}) $.
(2) $ RGEXn_{\beta}(X_{1})- RGEXn_{\beta}(X_{2})\geq \frac{1}{\beta}\left(\mu_{1}-\mu_{2}\right) $, where $ \mu_{1} = \int_{0}^{\infty} \bar{F}_{1}(x) \, dx $, and $ \mu_{2} = \int_{0}^{\infty} \bar{F}_{2}(x) \, dx $.
Proof. $ 1) $ Let $ \bar{F}_{1}(x)\leq \bar{F}_{2}(x) $, and since $ e^{1-\bar{F}^{\beta}(x)}\geq 1 $, for all $ \beta > 0 $. Then, the result follows.
$ (2) $ Since $ \bar{F}_{1}(x)\leq\bar{F}_{2}(x) $, then $ \bar{F}_{1}^{\beta}(x)\leq\bar{F}_{2}^{\beta}(x) $, for all $ \beta > 0 $, or equivalently $ e^{1-\bar{F}_{1}^{\beta}(x)}\geq e^{1-\bar{F}_{2}^{\beta}(x)} $. Therefore, we have
$ RGEXnβ(X1)−RGEXnβ(X2)=1β∫∞0[ˉF1(x)(e1−ˉFβ1(x)−1)−ˉF2(x)(e1−ˉFβ2(x)−1)]dx≥1β(∫∞0(ˉF1(x)−ˉF2(x))dx)=1β(μ1−μ2). $ |
Example 2.2. Suppose that two random variables that are continuously, $X_{1}$ and $X_{2}$, following $ Ud(0, t_{1}) $ and $ Ud(0, t_{2}) $ distributions with the CDFs $ F_{1}(x) = \frac{x}{t_{1}} $, $ 0\leq x\leq t_{1} $, and $ F_{2}(x) = \frac{x}{t_{2}} $, $ 0\leq x\leq t_{2} $, respectively. Moreover, the means are $ \mu_{1} = \frac{t_{1}}{2} $ and $ \mu_{2} = \frac{t_{2}}{2} $. If we let $ t_{2}\geq t_{1} $, then we have $X_{1} \leq_{\text{UstOr}} X_{2}$. With $ \beta = 1 $, we obtain
$ RGEXn1(X1)=t1(e−52)≤t2(e−52)=RGEXn1(X2), $ |
and
$ RGEXn1(X1)−RGEXn1(X2)=(e−52)(t1−t2)≥12(t1−t2)=μ1−μ2, $ |
where $ \left(t_{1} -t_{2}\right)\leq 0 $. Which assures the results in Theorem 2.2.
Theorem 2.3. Assume that two random variables that are continuously, $X_{1}$ and $X_{2}$, following distribution functions that are strictly increasing, ($F_{1}$ and $F_{2}$, respectively). If $X_{1} \leq_{\text{DisOr}} X_{2}$, and
$ RGEXn_{\beta}(X_{1}) = RGEXn_{\beta}(X_{2}), $ |
for a fixed $\beta > 0$. Consequently, up to a location parameter, the distributions of $X_{1}$ and $X_{2}$ are identical.
Proof. Suppose that $X_{1} \leq_{\text{DisOr}} X_{2}$ (i.e., the function $F_2^{-1}(v) - F_1^{-1}(v)$ is decreasing in $v$), and $ RGEXn_{\beta}(X_{1}) = RGEXn_{\beta}(X_{2}) $. Then, by a change of variable $v = F(x)$, we observe that (according to the equality given)
$ RGEXn_{\beta}(X_{2})-RGEXn_{\beta}(X_{1}) = \frac{1}{\beta} \int_0^1 \big((1 - v) \left(e^{1-(1-v)^{\beta}}-1\right) \big) d\big[F_{2}^{-1}(v) - F_{1}^{-1}(v)\big] = 0, $ |
for a fixed $\beta > 0$. Since $X_{1} \leq_{\text{DisOr}} X_{2}$, we are aware of that $F_{2}^{-1}(v) - F_{1}^{-1}(v)$ is a function considered to be decreasing of $v$. We now assert that for every $0 \leq v \leq 1$, $F_{2}^{-1}(v) - F_{1}^{-1}(v) = c$ (constant). Suppose, to the contrary, that there exists a subinterval $(\theta_{1}, \theta_{2})$ within $[0, 1]$ where $F_{2}^{-1}(v) - F_{1}^{-1}(v)$ fails to remain constant across $(\theta_{1}, \theta_{2})$. In this case,
$ 0=1β∫10((1−v)(e1−(1−v)β−1)d[F−12(v)−F−11(v)]≥1β∫θ2θ1((1−v)(e1−(1−v)β−1)d[F−12(v)−F−11(v)]>0, $ |
which is a contradiction. Consequently, for all $0 \leq v \leq 1$, $F_{2}^{-1}(v) - F_{1}^{-1}(v) = c$ (constant), indicating that $X_{1}$ and $X_{2}$ have an equal distribution according to the location parameter. Alternatively, in more detail, the proof can be argued by contradiction. Suppose that $F_2^{-1}(v) - F_1^{-1}(v)$ is not constant on some subinterval $(\theta_1, \theta_2)$. Then, because $F_2^{-1}(v) - F_1^{-1}(v)$ is decreasing, the integral over $(\theta_1, \theta_2)$ will be positive:
$ \frac{1}{\beta} \int_{\theta_1}^{\theta_2} (1 - v) \left(e^{1-(1-v)^{\beta}} - 1\right) d\left[F_2^{-1}(v) - F_1^{-1}(v)\right] > 0. $ |
This contradicts the equality $RGEXn_{\beta}(X_1) = RGEXn_{\beta}(X_2)$, implying that $F_2^{-1}(v) - F_1^{-1}(v)$ must be constant for all $v \in [0, 1]$.
Remark 2.1. As a deeper explanation of Theorem 2.3, the phrase "Consequently, up to a location parameter, the distributions of $X_1$ and $X_2$ are identical" means that the distributions of $X_1$ and $X_2$ are the same except for a shift or translation along the real number line. In other words, $X_1$ and $X_2$ have the same shape and structure in their distributions, but one is a shifted version of the other. For example, let $X_1$ and $X_2$ be $ Exp(\gamma) $ distributed random variables with the rate parameter $\gamma > 0$. If $X_1$ and $X_2$ both follow the same exponential distribution with the rate parameter $\gamma$, then their CDFs are:
$ F_1(x) = 1 - e^{-\gamma x}, \quad F_2(x) = 1 - e^{-\gamma x}. $ |
Here, $X_1$ and $X_2$ are identically distributed, and there is no shift ($c = 0$). Suppose that $X_2$ is a shifted version of $X_1$ by a constant $c > 0$. In this case, the CDF of $X_2$ is:
$ F_2(x) = F_1(x - c) = 1 - e^{-\gamma (x - c)}, \quad x \geq c. $ |
Here, $X_2$ is the same as $X_1$ but shifted to the right by $c$. The distributions are identical up to the location parameter $c$.
The following assertion gives an alternate formula for the residual cumulative generalized exponential entropy of $ X $. The sequel uses this formulation, which is in respect of an expanding convex function, to derive a number of findings.
Lemma 2.2. The following is true for $ \beta>0$ if $ X $ represents an entirely constantly non-negativity random variable with the limiting mean $ \mu = \mathbb{E}(X) $:
$ RGEXn_{\beta}(X) \geq \frac{\psi_{\beta}(\mu)}{\beta}, $ |
with noting that the function $ \psi_{\beta}(\cdot) $ is given in (2.6).
Proof. Given that $ \psi_{\beta}(\cdot) $ is a function to be convex, Jensen's inequality may be used as $ E(\psi_{\beta}(X))\geq\psi_{\beta}(E(X)) $, which obtains the proof.
The following characteristics of stochastic order are useful for comparing risk measurements and are also obtained by using Proposition 2.3.
Proposition 2.4. For $ \beta > 0 $, it is true that if $ X_{1} $ and $ X_{2} $ are completely continuously non-negative random variables that correspond to $ X_{1} \leq_{\mathit{\text{IcxOr}}} X_{2} $, then
$ \psi_{\beta}(X_{1}) \leq_{\mathit{\text{IcxOr}}} \psi_{\beta}(X_{2}), $ |
where (2.6) defines the function $ \psi_{\beta}(\cdot) $. $ X_{1} \leq_{\mathit{\text{IcxOr}}} X_{2} $ specifically suggests
$ RGEXn_{\beta}(X_{1}) \leq RGEXn_{\beta}(X_{2}). $ |
Proof. According to Theorem 4.A.8 of Shaked and Shanthikumar [13], $ \psi_{\beta}(X_{1}) \leq_{\text{IcxOr}} \psi_{\beta}(X_{2}) $ is an function which considered to be increasing convex for $ \beta > 0 $ in the function $ \psi_{\beta}(\cdot) $. Specifically, using Eq (2.5) and the concept of rising convex order, we obtain $ RGEXn_{\beta}(X_{1}) \leq RGEXn_{\beta}(X_{2}) $.
Proposition 2.5. Assume that $ X_1, \ldots, X_m $ be $ m $ independent non-negativity absolutely continuous random variables with the collective CDF $ F $, and $ Z_1, \ldots, Z_m $ are another set of $ m $ independent non-negative continuous random variables with the collective CDF $ F^{*} $. If $ X_i \leq_{\mathit{\text{IcxOr}}} Z_i $ for $ i = 1, 2, \ldots, m $, then for all $ \beta > 0 $, we obtain
$ RGEXn_{\beta}\big(\max\{X_1, X_2, \ldots, X_m\}\big) \leq RGEXn_{\beta}\big(\max\{Z_1, Z_2, \ldots, Z_m\}\big). $ |
Proof. Given that $ X_i \leq_{\text{IcxOr}} Z_i $ for $ i = 1, 2, \ldots, m $, using Shaked and Shanthikumar's [13] Corollary 4.A.16, we get
$ \max\{X_1, X_2, \ldots, X_m\} \leq_{\text{IcxOr}} \max\{Z_1, Z_2, \ldots, Z_m\}. $ |
The result then follows directly from Proposition 2.3.
Proposition 2.6. For $ \beta > 0 $, the following is true if $ X_{1} $ and $ X_{2} $ are non-negative randomized variables that match $ X_{1} \leq_{\mathit{\text{HrOr}}} X_{2} $:
$ \frac{RGEXn_{\beta}(X_{1})}{E(X_{1})} \leq \frac{RGEXn_{\beta}(X_{1})}{E(X_{2})}. $ |
Proof. Shaked and Shanthikumar [13] determined that, assuming $ X_{1} \leq_{\text{HrOr}} X_{2} $, the function $ \psi_{\beta}(\cdot) $, described in (2.6), is an increasing function to be convex such that $ \psi_{\beta}(0) = 0 $:
$ \frac{E(\psi_{\beta}(X_{1}))}{E(X_{1})} \leq \frac{E(\psi_{\beta}(X_{2}))}{E(X_{2})}. $ |
Therefore, Proposition 2.4 completes the evidence.
In this subsection, we will examine the characterization of the residual cumulative generalized exponential entropy based on order statistics. Using the well-known Müntz-Szász theorem, we first review the idea of a full sequence of functions and a lemma; for further information, see [14,15,16].
Lemma 2.3. (Müntz-Szász theorem; see Higgins [16], pp, 95–96). On a limited interval $ (\theta_{1}, \theta_{2}) $, for a function which considered integrable $ \Theta(z) $, if
$ \int_{\theta_{1}}^{\theta_{2}} z^{t_i} \Theta(z) \, dz = 0, \quad i \geq 1, $ |
for nearly all $ z \in (\theta_{1}, \theta_{2}) $, $ \Theta(z) = 0 $, where $ \{t_i, i \geq 1\} $ is a sequence of positive integers that increasing strictly fulfilling
$ \sum\limits_{j = 1}^\infty \frac{1}{t_j} = +\infty. $ |
In functional analysis, the well-known Lemma 2.3 states that the set of values $ \{z^{t_1}, z^{t_2}, \dots: 1 \leq t_1 < t_2 < \dots\} $ constitutes a complete sequence. It is important to note that Galambos [17] presents a straightforward version of the Müntz-Szász theorem along with a proof (see Theorem AlI.3). Furthermore, the Müntz-Szász theory for $ \{\psi_{t_i}(z), t_i \geq 1\} $ was extended by Hwang and Lin [14], where $ \psi(z) $ is monotone and absolutely continuous on a range $ (\theta_{1}, \theta_{2}) $. We examine characterization using the first-order statistics in the following theorem. Let $ Z_1, \dots, Z_t $ be completely continuously non-negativity random variables with the routine PDF $ f $ and CDF $ F $, and let $ t $ be independent and distributed in an identical manner. The function that is considered to be the survival function of the first-order statistics is thus expressed as follows: $ \bar{F}_{1;t}(z) = [\bar{F}(z)]^t $, $ z \geq 0 $.
Theorem 2.4. Assume that $ X $ and $ Z $ are two completely continual, non-negativity random variables, each with a PDF of $ f $ and $ h $ and a CDF of $ F $ and $ H $. Then, if and only if $ F $ and $ H $ are members of the same distribution family, albeit, with a different scale and location, we have
$ \frac{RGEXn_{\beta}(X_{1;t})}{E(X_{1;t})} = \frac{RGEXn_{\beta}(Z_{1;t})}{E(Z_{1;t})}, $ |
for every $ t = t_k $, $ k \geq 1 $, and for a given $ \beta > 0 $, such that
$ \sum\limits_{k = 1}^\infty t_k^{-1} = \infty. $ |
Proof. The necessary condition is inessential. For the sufficiency condition, after letting $ v = \bar{F}(x) $, we realize that
$ E(X_{1;t}) = \int_{0}^{\infty} \bar{F}_{1;t}(x) \, dx = \int_{0}^{1} \frac{v^{t}}{f\left(\bar{F}^{-1}(v)\right)} \, dv, $ |
and that,
$ βRGEXnβ(X1;t)=∫10vt(e1−vtβ−1)f(ˉF−1(v))dv. $ |
Consequently,
$ \frac{RGEXn_{\beta}(X_{1;t})}{E(X_{1;t})} = \frac{RGEXn_{\beta}(Z_{1;t})}{E(Y_{1;t})}, $ |
is equivalent to
$ \frac{\int_{0}^{1} \frac{v^{t}\left(e^{1-v^{t\beta}}-1\right)}{f\left(\bar{F}^{-1}(v)\right)}\, dv} {\int_{0}^{1} \frac{v^{t}}{f\left(\bar{F}^{-1}(v)\right)} \, dv} = \frac{\int_{0}^{1} \frac{v^{t}\left(e^{1-v^{t\beta}}-1\right)}{h\left(\bar{H}^{-1}(v)\right)}\, dv} {\int_{0}^{1} \frac{v^{t}}{h\left(\bar{H}^{-1}(v)\right)} \, dv}. $ |
Therefore,
$ ∫10vt(e1−vtβ−1)[1f(ˉF−1(v))−1Ψh(ˉH−1(v))]dv,=∫10vtΘ(v)dv, $ |
where
$ Θ(v)=(e1−vtβ−1)[1f(ˉF−1(v))−1Ψh(ˉH−1(v))], $ | (2.7) |
$ \Psi = \frac{\int_{0}^{1} \frac{v^{t}}{f\left(\bar{F}^{-1}(v)\right)} \, dv}{\int_{0}^{1} \frac{v^{t}}{h\left(\bar{H}^{-1}(v)\right)} \, dv}. $ |
For every $ t = t_k, \; k \geq 1 $, the latter relation is hypothesized to exist, so that $ \sum_{k = 1}^{\infty} t_k^{-1} = \infty $. Applying the Müntz-Szász theorem to the whole sequence $ {v^{t}, t \geq 1} $ and Lemma 2.3, the relation (2.7) provides $ \Theta(v) = 0 $, or equivalently, $ f\left(\bar{F}^{-1}(v)\right) = \Psi\, h\left(\bar{H}^{-1}(v)\right) $, for every $ 0 < v < 1 $. We may observe that $ F^{-1}(v) = \Psi\, H^{-1}(v) + c $, for any $ 0 < v < 1 $ and a real constant $ c $, if we remember that $ \frac{d}{dv} \overline{F}^{-1}(v) = \frac{1}{f\left(\bar{F}^{-1}(v)\right)} $. By the same manner, in Psarrakos and Toomaj [18], the CDFs $ F $ and $ H $ are members of the same distribution family, with a different scale and location.
Theorem 2.5. Consider two completely continuous, non-negativity random variables, $ X $ and $ Z $, each with PDFs of $ f $ and $ h $ and CDFs of $ F $ and $ H $. For a change in location, $ F $ and $ H $ are members of the same distribution family, if and only if
$ RGEXn_{\beta}(X_{1;t}) = RGEXn_{\beta}(Z_{1;t}), $ |
for a fixed $ \beta > 0 $ and for every $ t = t_j $, $ j \geq 1 $, where $ \sum_{k = 1}^\infty \frac{1}{t_k} = \infty $.
Proof. For $ \Psi = 1 $, the proof is comparable to Theorem 2.4.
In this section, we will examine the expansion of the residual cumulative generalized exponential entropy and obtain some results. The definition of the residual cumulative generalized exponential entropy is:
$ RGEXn_{\beta}(X) = \frac{1}{\beta} \int_{0}^{\infty} \bar{F}(x) \left( e^{1 - \bar{F}^\beta(x)} - 1 \right) dx. $ |
Using the Taylor series expansion for $ e^{1 - \bar{F}^\beta(x)}-1 $, we have:
$ e1−ˉFβ(x)−1=(1−ˉFβ(x))+(1−ˉFβ(x))22!+(1−ˉFβ(x))33!+⋯=∞∑k=1(1−ˉFβ(x))kk!. $ |
For $\beta > 0$, $\bar{F}^\beta(x) \geq 0$ for all $x$, so $1 - \bar{F}^\beta(x)$ is finite and the series converges. Substituting into the integral:
$ RGEXn_{\beta}(X) = \frac{1}{\beta} \sum\limits_{k = 1}^\infty \frac{1}{k!} \int_{0}^\infty \bar{F}(x) \left( 1 - \bar{F}^\beta(x) \right)^k dx. $ |
For $(1 - \bar{F}^\beta(x))^k$, we use the binomial theorem to obtain
$ (1 - \bar{F}^\beta(x))^k = \sum\limits_{j = 0}^k \binom{k}{j} (-1)^j \bar{F}^{\beta j}(x). $ |
Substituting this back yields
$ RGEXnβ(X)=1β∞∑k=11k!k∑j=0(kj)(−1)j∫∞0ˉF1+βj(x)dx. $ | (3.1) |
For $\beta > 0$, the survival function $\bar{F}(x)$ typically decreases to 0 as $x \to \infty$, ensuring the convergence of $\bar{F}^{1 + \beta j}(x)$.
The residual or excess of $ Z $, assuming that it surpasses a threshold $ t $, is represented by $ Z_t = [Z - t \mid Z > t] $ if the random variable $ Z $ represents the lifespan of a component or a system. In contrast, $ [Z \mid W] $ often indicates a random variable with a similar distribution as $ Z $ conditional on $ W $. The PDF of $ Z_t $ is obtained as follows:
$ f(z \mid t) = \frac{f(z)}{\bar{F}(t)}, \quad z > t. $ |
The equation for the function of the average residual life of $ Z $ with a finite mean $ \mu $ can potentially be calculated as
$ M(t)=M(Z;t)=EZ>t[Z−t∣Z>t],t≥0, $ | (3.2) |
in this case, $ E_{Z > t} $ denotes the expectation of the residual PDF $ f(z \mid t) $. With $ Z_\beta $ in place of $ Z $, the function of the mean residual life of $ Z_\beta $, represented as $ M_\beta (t) $, may be found using (3.2). We note that the best choice under the quadratic loss function is the function of the mean residual life of $ Z_\beta $:
$ Qls(\delta, Z_\beta \mid t) = (Z_\beta - t - \delta)^2, \quad Z_\beta > t, $ |
for excess of prediction, i.e.,
$ \delta^\ast(t) = \arg \min\limits_\delta E_{Z_\beta > t}[Qls(\delta, Z_\beta \mid t)] = M_\beta(t), \quad \beta > 0. $ |
Asadi et al. [19] noted that $ M_\beta (t) $ is a risk local measure that depends on the threshold $ t $. The Bayes risk is its global risk, as
$ Br(M_\beta) = E_\Pi [M_\beta (t)], $ |
where $ \Pi(t) $ is the distribution of prior for the threshold $ t $.
Theorem 3.1. Let $ Z $ have the function of the mean residual life $ M $, and assume the baseline prior $ \Pi(t) = f(t) $. Then, the residual cumulative generalized exponential entropy can be expressed by the Bayes risk of $ M_\beta (t) $ as follows:
$ RGEXnβ(Z)=∞∑k=11k!k∑j=0(kj)(−1)jjBr(Mβj+1)=∞∑k=11k!k∑j=0(kj)(−1)jjE[Mβj+1(Z)]. $ | (3.3) |
Proof. Under the substitution $ \Pi(t) = f(t) $, and utilizing the theorem of Fubini, we can express the Bayes risk as
$ Br(Mα)=∫∞0Mα(t)Π(t)dt=∫∞0Mα(t)f(t)dt=∫∞0(∫∞tˉFα(z)dzˉFα(t))f(t)dt=∫∞0ˉFα(z)(∫z0f(t)dtˉFα(t)dt)dz=∫∞0ˉFα(z)(1α−1(ˉF−α+1(z)−1))dz=1α−1∫∞0(ˉF(z)−ˉFα(z))dz=1α−1(μ−∫∞0ˉFα(z)dz), $ |
where $ 1\neq\alpha > 0 $. Therefore, we can see that
$ ∫∞0ˉFα(z)dz=μ−(α−1)Br(Mα). $ | (3.4) |
Substituting from (3.4) in (3.1), we obtain
$ RGEXnβ(Z)=1β∞∑k=11k!k∑j=0(kj)(−1)j[μ−(βj)Br(Mβj+1)]=−∞∑k=11k!k∑j=0(kj)(−1)jjBr(Mβj+1), $ |
where the last line is obtained from noting that the inner summation:
$ \sum\limits_{j = 0}^k \binom{k}{j} (-1)^j = (1 - 1)^k = 0, \quad \text{for } k \geq 1. $ |
Theorem 3.2. Assume that $ Z $ is a non-negativity, exactly continually random variable following the PDF $ f(z) $. In this case
$ RGEXnβ(Z)=−∞∑k=11k!k∑j=0(kj)(−1)jj(E(Zβj+1)+E(gβj+1(Z))), $ |
where $ E(Z_{\beta}) = \int_{0}^{\infty}\bar{F}^{\beta}(z) \, dz $, $ g_{\beta}(v) = \int_{0}^{v} M'(u) \bar{F}^{\beta-1}(u)\, du $, and $ v > 0 $.
Proof. From (3.1), we can rewrite it as
$ RGEXnβ(Z)=−1β∞∑k=11k!k∑j=0(kj)(−1)jj[1βj(μ−∫∞0ˉF1+βj(z)dz)]. $ | (3.5) |
Let $ \eta(z) = \frac{f(z)}{\bar{F}(z)} $ be the function of the hazard rate. We then have the integration
$ ∫∞0M(z)η(z)ˉFβ(z)dz=∫∞0(∫∞zˉF(t)dtˉF2−β(z))f(z)dz=∫∞0ˉF(z)(∫z0f(t)ˉFβ−2(t)dt)dz=1β−1(μ−∫∞0ˉFβ(z)dz). $ | (3.6) |
By substituting (3.6) in (3.5), and using the result $ M(u) \eta(u) = 1+M'(u) $, we get
$ RGEXnβ(Z)=−1β∞∑k=11k!k∑j=0(kj)(−1)jj[∫∞0M(z)η(z)ˉFβj+1(z)dz]=−1β∞∑k=11k!k∑j=0(kj)(−1)jj[E(Zβj+1)+∫∞0M′(z)ˉFβj+1(z)dz]=−1β∞∑k=11k!k∑j=0(kj)(−1)jj[E(Zβj+1)+∫∞0M′(z)∫∞xf(u)[ˉF(z)]βjdudz]=−1β∞∑k=11k!k∑j=0(kj)(−1)jj[E(Zβj+1)+∫∞0f(u)∫u0M′(z)[ˉF(z)]βjdzdu], $ |
then the result follows.
This part examines the connection between the residual cumulative generalized exponential entropy and the wealth order excess, sometimes referred to as the spread right order. Examining the standard deviations of two distribution functions is usually the simplest method to compare their variability. However, Shaked and Shanthikumar [13] developed and thoroughly analyzed stochastic ordering and different transformations for comparing the variability, since numerical measurements alone may not always give adequate information. One of them is the order of excess wealth, which is used to evaluate spread. The wealth excess convert for a non-negativity random variable $ Z$ follows a CDF $ H$ and PDF $ h $ is given by (cf. Fernandez-Ponce et al. [20])
$ ΔZ(v)=∫∞H−1(v)¯H(z)dz=∫1v(1−q)⋅1h(H−1(q))dq=∫1v(H−1(q)−H−1(v))dq, $ |
with noting that $ H^{-1}(v) = \inf\{z: F(z)\geq v \}, \; v\in (0, 1) $, is the quantile function of $ H $, and $ dz = \frac{d}{dq} H^{-1}(q) \, dq = \frac{1}{h(H^{-1}(q))} \, dq $. Therefore, the difference $ H^{-1}(q) - H^{-1}(v) $ measures the excess above the threshold $ F^{-1}(v) $ at a level $ q $.
This function and the function of the mean residual life are also connected in this manner by the following connection:
$ MZ(F−1(u))=ΔZ(v)1−v,0<v<1. $ | (3.7) |
Equation (3.7) is used to prove the following theorem.
Theorem 3.3. If $ Z $ has a CDF of $ H $ and is a completely continuous, non-negative random variable, then
$ RGEXnβ(Z)=∞∑k=11k!k∑j=0(kj)(−1)jj[∫10MZ(H−1(v))(1−v)βjdv]. $ | (3.8) |
for all $ \beta > 0 $.
Proof. The residual cumulative generalized exponential entropy given in (3.3), can be rewritten as
$ RGEXnβ(Z)=∞∑k=11k!k∑j=0(kj)(−1)jjE[Mβj+1(Z)]=∞∑k=11k!k∑j=0(kj)(−1)jjβj+1E[MZ(Zβj+1)], $ |
and the result follows.
Example 3.1. (1) Suppose that $ Z $ has a distribution of $ Ud(0, d) $. It is clear to see that
$ MZ(F−1(v))=d(1−v)2. $ |
Consequently, using (3.8), we obtain
$ RGEXnβ(Z)=∞∑k=11k!k∑j=0(kj)(−1)jjd2[∫10(1−v)βj+1dv]=∞∑k=11k!k∑j=0(kj)(−1)jjd2(βj+2), $ |
$ \beta > 0 $.
(2) Let us examine the Pareto distribution, characterized by a scale parameter $ p > 0 $ and a shape parameter $ s > 0 $, where the function of survival is provided by $ \overline{H}(z) = \frac{p^{s}}{(z + p)^{s}} $ for $ z \geq 0 $. It is straightforward to observe that
$ MZ(F−1(v))=p(1−v)−1s(s−1). $ |
Consequently, using (3.8), we obtain
$ RGEXnβ(Z)=∞∑k=11k!k∑j=0(kj)(−1)jjp(s−1)[∫10(1−v)βj−1sdv]=∞∑k=11k!k∑j=0(kj)(−1)jjsp(s(βj+1)−1)(s−1), $ |
$ \beta > 0 $.
The residual cumulative generalized exponential entropy is estimated in this section using the empirical residual cumulative entropy. For any $ \beta > 0 $, and the random sample $X_1, X_2, \ldots, X_n $, the empirical estimation of the residual cumulative generalized exponential entropy is expressed as
$ RGEXnβ(ˆFn)=1β∫∞0ˆFn(x)[e1−ˆFβn(x)−1]dx=1βn−1∑j=1∫Xj+1;nXj;n(1−jn)[e1−(1−jn)β−1]dx=1βn−1∑j=1Ωj+1(1−jn)[e1−(1−jn)β−1], $ | (4.1) |
with noting that $ X_{1;n} \leq X_{2;n} \leq \ldots \leq X_{n; n} $ are the associated order statistics of the random sample, and the sample spacings are $ \Omega_{j+1} = X_{j+1;n}-X_{j; n} $, $ j = 1, 2, ..., n-1 $. For the sample that corresponds to $ F $, the empirical distribution function is described by $ \widehat{F}_{n}(x) = \sum_{j = 1}^{n-1}\frac{j}{n} \mathbb{A}_{[x_{j}, x_{j+1}]}(x), x\geq0 $, with the indicator function, $ \mathbb{A}_{\varpi}(x) = 1 $, $ x\in \varpi $. Moreover, with $ \beta > 0 $, we can use the expansion form of the residual cumulative generalized exponential entropy in (3.1) to present its empirical expression as follows:
$ RGEXnβ(ˆFn)=1β∞∑k=11k!k∑j=0(kj)(−1)j∫∞0ˉF1+βj(x)dx=1β∞∑k=11k!k∑j=0(kj)(−1)jn−1∑l=1Ωl+1(1−ln)1+βj. $ | (4.2) |
Utilizing the expansion form of the empirical residual cumulative generalized exponential entropy in (4.2), we can now provide a central limit theorem for this measure, which is derived from a random sample with an exponential distribution.
Theorem 4.1. A sample selected at random $ X_1, X_2, \ldots, X_n $ drawn from a common $ Exp(\gamma) $ distribution is considered. In this case,
$ RGEXnβ(ˆFn)−E[RGEXnβ(ˆFn)]√Var[RGEXnβ(ˆFn)]⟶standard normal distribution, $ |
where $ \beta > 0 $.
Proof. The empirically residual cumulative generalized exponential entropy measure can be written as a total of the independent exponential random variables $ X_{l} $, $ l = 1, 2, ..., n $, using the expansion (4.2), where the variance and expected value are given by (noting that the spacing $ \Omega_{l+1} $ are independent and distributed by $ Exp(\gamma (n - l)) $)
$ E[Xl]=1nγβ∞∑k=11k!k∑j=0(kj)(−1)j(1−ln)βj, $ | (4.3) |
and
$ Var[Xl]=1n2γ2β2[∞∑k=11k!k∑j=0(kj)(−1)j(1−ln)βj]2. $ | (4.4) |
Consider the following: $ \Phi_{l, q} = E[|X_{l}-E(X_{l})|^{q}] $, $ q = 2, 3 $. From (4.4), one can derive the following estimates for $ n $ considered to be large, as follows
$ n∑l=1Φl,2=n∑l=1E[|Xl−E(Xl)|2]=n∑l=1Var[Xl]=1n2γ2β2n∑l=1[∞∑k=11k!k∑j=0(kj)(−1)j(1−ln)βj]2≈1n2γ2β2∫10(e1−xβ−1)2dx=1n2γ2β2g2. $ |
Additionally, given whatever random variable $ X_l $ with an exponential distribution, the following result may be reached. From (4.3), we obtain (observing that $ E[|X_l - E(X_l)|^3] = \frac{2(6-e)[E(X_l)]^3}{e} $, see, [21,22])
$ n∑l=1Φl,3=2(6−e)en∑l=1[E(Xl)]3=2(6−e)e(nγβ)3n∑l=1[∞∑k=11k!k∑j=0(kj)(−1)j(1−ln)βj]3≈2(6−e)en3γ3β3∫10(e1−xβ−1)3dx=2(6−e)en3γ3β3g3. $ |
Taking note of that
$ gq=∫10(e1−xβ−1)qdx,q=2,3, $ |
and the integrand $ (e^{1-x^{\beta}}-1)^{q} $ is bounded and continuous over the interval $ [0, 1] $. Therefore, it is true for large $ n $, given an adequate function $ Gn $, that
$ ∑nl=1Φl,3∑nl=1Φl,2≈Gnn⟶n→∞0. $ |
Thus, the central limit theorem's Lyapunov condition is satisfied (see, for example, [23]), thus completing the proof.
In this subsection, a different non-parametric estimator can be developed as follows. The residual cumulative generalized exponential entropy given in (3.1) can be rewritten as
$ RGEXnβ(X)=1β∞∑k=11k!k∑j=0(kj)(−1)j∫∞0ˉF1+βj(x)dx=1β(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)(∫∞0xf(x)ˉFβj(x)dx)). $ | (4.5) |
The residual cumulative generalized exponential entropy was introduced as an L-functional by Zardasht [24]. Similarly, $ RGEXn_{\beta}(X) $ in (4.5) can be represented as
$ RGEXnβ(X)=1β(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)(∫∞0xˉFβj(x)dF(x)))=1β(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)(∫∞0xLnβj(F(x))dF(x))), $ | (4.6) |
where $ Ln_{\beta j}(v) = (1-u)^{\beta j} $, $ 0\leq u\leq1 $. In the follow-up, we can produce an estimate for $ RGEXn_{\beta}(X) $ using the following L-statistic by replacing $ F $ in (4.6) with $ \widehat{F}_{n} $:
$ RGEXnβ∗(ˆFn)=1β(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)(∫∞0xLnβj(ˆFn)dˆFn))≈1β(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)n(n∑l=1Xl;nLnβj(ln))). $ | (4.7) |
Example 4.1. A random sample $ X_{1}, X_{2}, \dots, X_{n} $ selected from the $ EXP(1) $ distribution is considered. $ \Omega_{l+1} = X_{(l+1)} - X_{(l)} $ provides the sample spacing for $ l = 1, 2, \dots, n-1 $, which are independent. Each $ \Omega_{j+1} $ has an exponential distribution with a parameter of $ (n - l) $. Thus, from (4.1), we obtain the following:
(1) The mean of $ RGEXn_{\beta}(\widehat{F}_{n}) $ is
$ E[RGEXnβ(ˆFn)]=1nβn−1∑l=1(e1−(1−ln)β−1). $ |
(2) The variance of $ RGEXn_{\beta}(\widehat{F}_{n}) $ is
$ Var[RGEXnβ(ˆFn)]=1n2β2n−1∑l=1(e1−(1−ln)β−1)2. $ |
It is possible to infer, from Eq (4.7) and the relations (4.6.6)–(4.6.8) provided by Arnold et al. [25], that
(1) The mean of $ RGEXn_{\beta}^{*}(\widehat{F}_{n}) $ is
$ E[RGEXn∗β(ˆFn)]=1β∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)nn∑l=1Lnβj(ln)l∑p=11n−p+1. $ |
(2) The variance of $ RGEXn_{\beta}^{*}(\widehat{F}_{n}) $ is
$ Var[RGEXn∗β(ˆFn)]=1β2(n∑l=1((∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)nLnβj(ln))2l∑p=11(n−p+1)2)+2n∑l1=1n∑l2=1(∞∑k=11k!k∑j=0(kj)(−1)j(1+βj)nLnβj(l1n))(∞∑k=11k!k∑j=0(kj)×(−1)j(1+βj)nLnβj(l2n))l1∑p=11(n−p+1)2). $ |
Remark 4.1. The intervals of confidence for $ RGEXn_{\beta}(X) $ can possibly be computed using the results of Theorem 4.1 if the random variables have an exponential distribution. In particular, from (4.1) and (4.7), it is true for any specific $ \beta > 0 $ that
$ RGEXnβ(ˆFn)±χδ2√Var[RGEXnβ(ˆFn)], $ |
$ RGEXn∗β(ˆFn)±χδ2√Var[RGEXn∗β(ˆFn)], $ |
where the critical point of the standard normal distribution at $ \frac{\delta}{2} $ is shown by $ \chi_{\frac{\delta}{2}} $.
The mean and variance of the empirical residual cumulative generalized exponential function of entropy for the aforementioned relationships are shown in Table 1. Sample sizes of $ 10, 20, 30, 40 $, and $ 50 $ were chosen, with varying values of the order $ \beta $. The residual cumulative generalized exponential entropy's precise values are computed as follows: $ RGEXn_{1}(X) = 0.718282 $, $ RGEXn_{2}(X) = 0.515039 $, and $ RGEXn_{3}(X) = 0.398348 $. On the basis of the results in Table 1, we can conclude the following
Sample size | β | $ E[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ E[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ |
n = 10 | 1 | 0.633799 | 0.0618118 | 0.636254 | 0.086091 |
2 | 0.471249 | 0.0302985 | 0.57848 | 0.0651343 | |
3 | 0.368875 | 0.0174016 | 0.530634 | 0.056318 | |
n = 20 | 1 | 0.675683 | 0.0343049 | 0.027132 | 0.617761 |
2 | 0.493352 | 0.0160719 | 0.55787 | 0.0203291 | |
3 | 0.38382 | 0.00911087 | 0.507678 | 0.017622 | |
n = 30 | 1 | 0.689803 | 0.023654 | 0.611674 | 0.0135907 |
2 | 0.500628 | 0.0109196 | 0.550958 | 0.0101643 | |
3 | 0.388709 | 0.00616504 | 0.499906 | 0.0061073 | |
n = 40 | 1 | 0.696893 | 0.0180386 | 0.608645 | 0.00826977 |
2 | 0.504248 | 0.0082666 | 0.547493 | 0.00618269 | |
3 | 0.391136 | 0.00465795 | 0.495995 | 0.00438061 | |
n = 50 | 1 | 0.701156 | 0.014575 | 0.606833 | 0.00560691 |
2 | 0.506414 | 0.00665019 | 0.545411 | 0.00419222 | |
3 | 0.392587 | 0.00374276 | 0.493641 | 0.00365316 |
$ (1) $ It is evident that the mean converges to the true value, and the variability of the empirical measurement approaches zero as the sample size increases.
$ (2) $ For any fixed $ n $ and increasing $ \beta $, the variance decreases.
$ (3) $ For any large $ n $, the second estimator provides a more accurate result (by decreasing the variance) compared with the first.
Example 4.2. Reaven and Miller [26] investigated the connection between insulin and blood chemistry indicators of glucose tolerances in 145 non-fat individuals. They visualized the data in three dimensions using the Stanford Accelerator Linear Center's PRIM9 technology and found an odd pattern that resembled a big blob with two wings pointing in separate directions. Three categories were created from the 145 observations: Overt diabetes, Chemical diabetics, and Normals. Additionally, the 145 observations were divided into three groups: 33 for overt diabetes, 36 for chemical diabetes, and 76 for normal diabetes. Five factors for every single individual were examined as follows:
(1) Relative of weight ($ \Lambda_{1} $),
(2) Test plasma glucose level ($ \Lambda_{2} $),
(3) Fasting plasma glucose level ($ \Lambda_{3} $),
(4) Plasma insulin during the test ($ \Lambda_{4} $), and
(5) Steady state plasma glucose ($ \Lambda_{5} $).
For the $\Lambda_{5}$ data set, we fit it to the exponential distribution with the parameter $\gamma = 0.00433$. Figure 2 shows the histogram of the $\Lambda_{5}$ data set with the exponential curve, along with the empirical and theoretical CDFs. To statistically validate this fit, we performed a Kolmogorov-Smirnov test, which yielded a p-value of 0.051032. This result supports the suitability of the $EXP(0.00433)$ distribution for modeling these data.
Figure 3 shows the theoretical residual cumulative generalized exponential entropy, which was computed using this exponential parameter distribution and the confidence interval (at $ \delta = 0.05 $) obtained from Eq (4.1). It is clear that the estimators' confidence intervals contain the theoretical value.
In this section, we will apply some properties and applications with the generalized exponential entropy including symmetry characterization and classification problem with pattern of recognition.
Using (1.5), we can define the continuous case of the generalized exponential entropy of the continual random variable $ Z $ with the PDF $ f $ as follows:
$ GEXn(Z)=1β∫∞−∞f(z)(e1−fβ(z)−1)dz, $ | (5.1) |
where $ \beta\in \mathbb{R}\backslash\{0\} $. Several interesting features of the extended exponential entropy of order statistics appear when the PDF of the underlying identical besides the independent of random variables is symmetric. With an underlying distribution $ Z $ containing the $ lth $-order statistic $ Z_{l; t} $, $ 1\leq l\leq t $, the PDF of a sample of size $ t $ is derived by
$ fl;t(z)=1Betaf(l,t−l+1)Fl−1(z)¯Ft−l(z)f(z), $ | (5.2) |
with nothing that $ Beta_{f}(l, t-l+1) = \frac{\Gamma(l)\Gamma(t-l+1)}{\Gamma(t+1)} $. We begin with two lemmas discussed by Fashandi and Ahmadi [27] and Balakrishnan and Selvitella [28], respectively, the definition of $ f_{r; t} $ in (5.2) and the symmetry assumption serve as the immediate foundation for the proof.
Lemma 5.1. [27] The following result is supported by $ G_Z $, PDF $ f $, and CDF $ F $, and $ Z $ is a continuous random variable as
$ f\left(F^{-1}(v)\right) = f\left(F^{-1}(1 - v)\right) \quad \mathit{\text{for all}} v \in (0, 1), $ |
which suggests the symmetry of $ F(z) $ with respect to a constant $ g_{n} \in G_Z $.
Lemma 5.2. [28] Assume that the parent distribution of the order statistic $ Z_{l; t} $, $ l = 1, ..., t $, has a PDF $ f $ with noting $ f(\mu + z) = f(\mu-z) $, $ z\geq 0 $. We proceed with the following analysis:
$ F(\mu + z) = \overline{F}(\mu-z), f_{l;t}(\mu + z) = f_{t-l+1;t}(\mu-z). $ |
Theorem 5.1. Let $ Z_{1}, ..., Z_{t} $ be identical, including independent distributed observations over $ Z $ whose PDF is regarded as symmetric around its mean $ \mu $. Consequently, we have
$ (1) $ In the event that $ t $ is deemed to be odd, $ GEXn_{\beta}(Z_{l; t}) = GEXn_{\beta}(Y_{t-l+1;t}) $, $ l = 1, ..., t $.
$ (2) $ If and only if $ GEXn_{\beta}(Z_{1;t}) = GEXn_{\beta}(Z_{t; t}) $, $ \forall t\geq 1 $, then $ Z $ has a symmetric PDF.
Proof. $ (1) $ Lemma 5.2 and Eq (5.1) provide us with
$ GEXnβ(Zl;t)=1β∫∞−∞fl;t(z)(e1−fβl;t(z)−1)dz1β∫∞−∞fl;t(μ+z)(e1−fβl;t(μ+z)−1)dz=1β∫∞−∞ft−l+1;t(μ−z)(e1−fβt−l+1;t(μ−z)−1)dz=1β∫∞−∞ft−l+1;t(z)(e1−fβt−l+1;t(z)−1)dz=GEXnβ(Zt−l+1;t). $ |
$ (2) $ The first part of this theorem establishes the necessity. We now turn to the sufficiency. Suppose that $ GEXn_{\beta}(Z_{l; t}) = GEXn_{\beta}(Z_{t; t}) $ holds for all $ t \geq 1 $. Using Lemma 5.1, we derive the following, for every $ v \in (0, 1) $,
$ f(F^{-1}(1-v)) = f(F^{-1}(v)), $ |
which leads to $ -\frac{d}{du}F^{-1}(1-v) = \frac{d}{dv}F^{-1}(v) $. Consequently, $ -F^{-1}(1-v) = F^{-1}(v) + g_{n} $, and thus $ f(-F^{-1}(v) - g_{n}) = f(F^{-1}(v)) $, where $ g_{n} $ is a constant, which is valid for all $ v \in (0, 1) $. Substituting $ F^{-1}(v) = -\frac{g_{n}}{2} + z $, we obtain $ f(-\frac{g_{n}}{2} - z) = f(-\frac{g_{n}}{2} + z) $ for all $ z \in \mathbb{R} $, thereby completing the proof of the theorem.
Corollary 5.1. In alignment with Theorem 5.1, let $ \Delta GEXn_{\beta}(Z_{p; t}) = \; GEXn_{\beta}(Z_{p+1;t})-GEXn_{\beta}(Z_{p; t}) $ represent the forward difference operator with respect to $ p $, where $ 1 \leq p \leq t-1 $. It follows that $ \Delta GEXn_{\beta}(Z_{l; t}) = -\Delta GEXn_{\beta}(Z_{t-l; t}) $, for $ l = 1, ..., t $.
Remark 5.1. Define $ \Xi_{t} $ as $ GEXn_{\beta}(Z_{1;t})-GEXn_{\beta}(Z_{t; t}) $. The condition $ \Xi_{t} = 0 $, for $ t = 1, 2, ... $, holds if and only if $ Z $ exhibits symmetry. Consequently, $ \Xi_{t} $ can serve as a fundamental measure of symmetry and as a statistic for testing symmetry.
From the assumptions outlined in Corollary 5.1, it can be deduced that the fractional generalized entropy $ GEXn_{\beta}(Z_{l; n}) $ attains either a local maximum at the median. This property is illustrated using the $ Ud(-1, 1) $ distribution. Specifically, for the generalized exponential entropy of the median $ (l = 4) $ with $ t = 7 $, the maximum values are $ -0.7269227 $ for $ \beta = 1 $, $ -0.56026 $ for $ \beta = 2 $, $ -0.4016726 $ for $ \beta = 3 $, and $ -0.3036904 $ for $ \beta = 4 $ (refer to Figure 4).
The probabilistic measures of information in the classification problems using pattern recognition will be covered in this section. For classification problems, uncertainty measurements are a helpful tool; for instance, one might refer to [29] and [30] in this context. We will use the data presented in Example 4.2 to discuss its relevant pattern of recognition using the generalized exponential entropy given in (1.5). The variables and their correlation are presented in Figure 5.
As indicated in Table 2(I), we select 30 samples for each data categorization in order to build an interval number model. We next identify a sample that has both the greatest and lowest values. An anonymous test case of every possible occurrence in the dataset is displayed. Assuming that the selected singleton data sample (1.18,108,486,297,220) originates from the chemical diabetic group (a singleton is a quantity that occurred only once in a dataset). To identify the singletons in a sample, we must first select a sample from a sequence of data and then note the occurrences of the values in that sample.
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
Normal | [0.74, 1.2] | [74,112] | [269,418] | [81,267] | [29,273] |
Chemical diabetic | [0.83, 1.2] | [75,114] | [413,643] | [109,748] | [60,300] |
Overt diabetic | [0.74, 1.2] | [120,353] | [538, 1520] | [10,460] | [150,458] |
(II) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \mathbb{P}(\text{normal}) $ | 0.319397 | 0.45749 | 0.318917 | 0.395996 | 0.329123 |
$ \mathbb{P}(\text{chemical diabetic}) $ | 0.361206 | 0.483345 | 0.603506 | 0.234745 | 0.405461 |
$ \mathbb{P}(\text{overt diabetic}) $ | 0.319397 | 0.0591652 | 0.0775768 | 0.369259 | 0.265417 |
Subsequently, we create five distinct probability distributions using Kang et al.'s [31] technique, which is based on the closeness between interval numbers. $ R_{1} = [\lambda_{1}, \lambda_{2}] $ and $ R_{2} = [\lambda^{*}_{1}, \lambda^{*}_{2}] $ are the two ranges taken into account. The distance between the ranges $ R_{1} $ and $ R_{2} $ is then calculated by
$ I(R1,R2)=[(λ1+λ22)−(λ∗1+λ∗22)]2+13[(λ1−λ22)2+(λ∗1−λ∗22)2]. $ |
Furthermore, their similarity $ \rho(R_{1}, R_{2}) $ is explained as
$ ρ(R1,R2)=11+ζI(R1,R2), $ |
in which $ \zeta $ is the supporting coefficient; one example of its application is to set $ \zeta $ to 5. For range the $ R_{1} $, we use the ranges listed in Table 2(I); for the range $ R_{2} $, we use individual values from the selected sample to generate the given probability distributions (for example, the range of for the value 1.18 in the $ \Lambda_{1} $ attribute is $ R_{2} = [1.18, 1.18] $). Table 2(II) shows that each of the five evaluated criteria produces three similarity values. A probability distribution is then created using the normalized representation of this data. These probability distributions are then evaluated using our generalized exponential entropy measure (with $ \beta = 1 $, $ \beta = 2 $, and $ \beta = 3 $) and are shown in Table 3(I). We also use $ W^{*}(T) = e^{-T} $ as the weighting foundation due to the monotonicity of the function that is considered to be exponential. The weights are then obtained by normalizing them. For example, when the generalized exponential entropy's $ \Lambda_{3} $ characteristic is used, the procedure produces
$ W∗(Λ3)=e−GEXn(Λ3)e−GEXn(Λ1)+e−GEXn(Λ2)+e−GEXn(Λ3)+e−GEXn(Λ4)+e−GEXn(Λ5), $ |
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \beta=1 $ | 0.945856 | 0.748902 | 0.722495 | 0.922879 | 0.931814 |
$ \beta=2 $ | 0.71489 | 0.604572 | 0.5662 | 0.699949 | 0.705139 |
$ \beta=3 $ | 0.539139 | 0.488135 | 0.4556 | 0.531995 | 0.534171 |
(II) Item | $ W^{*}(\Lambda_{1}) $ | $ W^{*}(\Lambda_{2}) $ | $ W^{*}(\Lambda_{3}) $ | $ W^{*}(\Lambda_{4}) $ | $ W^{*}(\Lambda_{5}) $ |
$ \beta=1 $ | 0.181642 | 0.221183 | 0.227102 | 0.185864 | 0.18421 |
$ \beta=2 $ | 0.188615 | 0.210614 | 0.218853 | 0.191454 | 0.190463 |
$ \beta=3 $ | 0.194114 | 0.204272 | 0.211027 | 0.195506 | 0.195081 |
The weighted values $ W^{*}(\Lambda_{t}) $, $ t = 1, 2, 3, 4, 5 $, corresponding to the five characteristics, are presented in Table 3(II). Therefore, the final probability distribution of the generalized exponential entropy measure is listed as:
$ (1) $ Under $ \beta = 1 $, we obtain
$ P(normal)=0.36586,P(chemical diabetic)=0.427895,P(overt diabetic)=0.206244. $ |
$ (2) $ Under $ \beta = 2 $, we obtain
$ P(normal)=0.364894,P(chemical diabetic)=0.424175,P(overt diabetic)=0.210931. $ |
$ (3) $ Under $ \beta = 3 $, we obtain
$ P(normal)=0.364377,P(chemical diabetic)=0.421197,P(overt diabetic)=0.214426. $ |
Following that, it was concluded that the chosen sample is most likely to be in the chemical diabetic category. As a result, in this case, the conclusion was correct.
Using this strategy, we examined all 145 samples via the entropy and generalized exponential entropy approaches over a range of $ \beta $ values, including 33 in the overt diabetic, 36 in the chemical diabetic, and 76 in the normal categories. The recognition rates are displayed in Table 4, where the entropy technique yields 72.03%. In the meanwhile, 59.23 is obtained using the extended exponential entropy technique. Moreover, when $ \beta = 1 $, we can see the probabilities when we choose the 76 normal categories tested samples in Figure 6, the 36 chemical diabetic categories tested samples in Figure 7, and the 33 overt diabetic categories tested samples in Figure 8 (the higher probability indicates the right choice).
Method | Normal | Chemical diabetic | Overt diabetic | In general |
Entropy method | 100% | 55.5% | 60.6% | 72.03% |
Generalized exponential entropy | ||||
method | 100% | 44.4% | 33.3% | 59.23% |
It must be emphasized that the results obtained in this section are based on the specific dataset used in our analysis and should not be interpreted as a general statement of preference. The performance of the proposed model may vary with different datasets, where its advantages could become more evident.
In this study, we emphasized that the study of the continuous case must be carried out and not just the discrete case, as its importance appeared in many of the applications used, such as dealing with the order statistics, as dealing with them in the discrete case is not flexible and limited. In addition, it is important to study the model based on the distribution function because of its many advantages. Besides, we have seen that the residual cumulative generalized exponential entropy measure is a generalization of the original model presented by Rao et al. [2]; this is one of the reasons why we do not rely on the original model. Moreover, proving that this measure is bounded contributed to solving some problems that have appeared in some theories and made it flexible in dealing with them. In addition, the measure's reliance on the exponential function made us use its expansion in studying some topics, like the order of excess wealth, Bayes risk, and estimations. Moreover, the real data were selected to see the application of this measure in terms of non-parametric estimators and the extent of suitability of these estimators with the proposed measure. On the other hand, this data was used to solve the problem of classification, as is clear at the end in Table 4, where the extent of the efficiency of using this measure to solve the problem at hand was shown.
Overall, the implications of the residual cumulative generalized exponential entropy as an extension of the residual cumulative entropy, which tends to it when $ \beta\rightarrow 0 $, has been presented. Numerous findings have been examined, including non-negativity, limits, relationship to the measure of classical differential measure of entropy, and preservation features, with a few well-known and familiar stochastic comparisons. Besides, some of those features are verified with some well-known distributions. In addition, some characterization of our model based on the first-order statistics has been obtained. Moreover, under the Taylor series expansion, some results on the expansion of the residual cumulative generalized exponential entropy, such as Bayes risk and the connection with the transform of excess wealth, have been explained. On top of that, we have examined the issue of using its empirical CDF to estimate the residual cumulative generalized exponential entropy. In this context, we estimate this measure using two distinct empirical estimators of the CDF. A theorem of the centralized limitation for the empirical measurement of randomly samples drawn from a distribution that is considered to be exponential is developed for the first estimator. A theorem of the central limit solution for empirical measuring constructed from a randomly sample using an unknown distribution is provided; however, it is also provided for the second estimator. Both methods were used on the data, and we found that they are close in their results, as the average values give results close to the true values, but there is some slight advantage for the second estimator. Moreover, the continuous case of generalized exponential entropy is discussed to illustrate the symmetry characterization of order statistics, using an example of a symmetric uniform distribution that shows that the median is the point of symmetry. On the other hand, generalized exponential entropy has been discussed in many areas, such as multi-criteria decision-making. Therefore, we have applied this model to the classification issue by utilizing the pattern recognition of a diabetes dataset and comparing it with the classical entropy, which shows superiority to the classical entropy measure.
In future work, we can implement the obtained measure to different topics like the concomitants of order statistics and tests of hypotheses and compare it against other existing entropy-based models in terms of interpretability, efficiency, or computational feasibility. Moreover, we aim to extend our analysis to additional common distributions, such as the normal and gamma distributions, which currently require numerical solutions. Investigating these distributions in greater detail will help illustrate the broader applicability of the measure and provide further insights into its practical utility. Furthermore, while the current study relies on closed-form expressions for the variance and mean (which eliminate the need for simulation-based data), future work will incorporate simulation studies to compute the mean square error. This will allow us to further validate the estimator's accuracy and assess the convergence of bias as the sample sizes increase. Moreover, we plan to explore the integration of the stochastic precedence order to evaluate its potential contributions and impact on our theoretical framework, thereby broadening the scope of stochastic ordering relations analyzed in this study.
The authors declare they have not used artificial intelligence (AI) tools in the creation of this article.
This study is supported via funding from Prince Sattam bin Abdulaziz University project number (PSAU/2025/R/1446).
The authors declare there is no conflict of interest.
The following Mathematica codes are provided to allow readers to reproduce the results presented in the paper.
Beta = 2; Gamma = 1;
fx = 1 - Exp[-Gamma x];
RGEXn = N[1/Beta Integrate[(1 - fx) (Exp[1 - (1 - fx)^Beta] - 1),
{x, 0, Infinity}]];
n = 10;
mean = N[1/(n Beta) Sum[(Exp[1 - (1 - l/n)^Beta] - 1), {l, 1, n - 1}]];
var = N[1/(n Beta)^2 Sum[(Exp[1 - (1 - l/n)^Beta] - 1)^2,
{l, 1, n - 1}]]
y = Sort[{data}];
n = Length[y];
Beta = Beta;
Gamma = 0.00433;
fx = 1 - Exp[-Gamma x];
RGEXn = N[1/Beta Integrate[(1 - fx) (Exp[1 - (1 - fx)^Beta] - 1),
{x, 0, Infinity}]];
s1 = N[1/Beta Sum[(y[[i + 1]] - y[[i]]) (1 - i/n)
(Exp[1 - (1 - i/n)^Beta] - 1), {i, 1, n - 1}]];
var1 = N[Variance[y]];
c1 = s1 + 1.96 Sqrt[var1];
c2 = s1 - 1.96 Sqrt[var1];
Plot[{STMn, c1, c2}, {Beta, 0.001, 3},
PlotLegends -> {"RGEXn", "Upper", "Lower"},
AxesLabel -> Automatic,
PlotStyle -> {Black, Red, {Dashed, Red, AbsoluteThickness[3]}},
Frame -> True]
rm(list = ls())
# Load necessary libraries
library(ggplot2)
# Define parameters
Theta < - 4
n < - 7
a < - -1
b < - 1
# Define the function for fyos1
fyos1 < - function(y, r) {
ffy < - (y - a) / (b - a)
fy < - 1
return((gamma(n + 1) * ffy^(r - 1) * (1 - ffy)^(n - r) * fy) /
(gamma(r) * gamma(n - r + 1)))
}
# Define the generalized function GF
GF < - function(r) {
integrand < - function(y) {
fyos1_val < - fyos1(y, r)
return(fyos1_val*(exp(1-(fyos1_val)^(Theta))-1))
}
result < - integrate(integrand, a, b)$value
return((1 / Theta) * result)
}
# Create the plot data
r_values < - 1:n GF_values < - sapply(r_values, GF)
# Create a data frame for ggplot
df < - data.frame(r = r_values, GF = GF_values)
# Plot using ggplot2
ggplot(df, aes(x = r, y = GF)) +
geom_point() +
geom_line() +
labs(x = "l", y = "", title = expression(paste(beta, " = 3"))) +
theme_minimal(base_size = 15)
# Clear the environment
rm(list = ls())
library(heplots)
library(GGally)
data("Diabetes")
# Select only the numerical variables
df < - Diabetes[, c("relwt", "glufast", "glutest", "instest", "sspg")]
# Create a scatterplot matrix
ggpairs(df, lower = list(continuous = "smooth"))
[1] | WHO (2010) World Health Organization. Developing sexual health programmes: a framework for action. Geneva. Available from: http://apps.who.int/iris/bitstream/10665/70501/1/WHO_RHR_HRP_10.22_eng.pdf. |
[2] | UNFPA (2010) Sexual and Reproductive Health For All. Reducing Poverty, Advancing Development and Protecting Human Rights. Available from: http://www.unfpa.org/publications/sexual-and-reproductive-health-all#sthash.xZTrIzAD.dpuf: UNFPA. |
[3] | WHO (2006) World Health Organization. Defining Sexual Health, Report of Technical Consultation on Sexual Health, January 2002, Geneva. Available form: http://www.who.int/reproductivehealth/publications/sexual_health/defining_sexual_health.pdf. |
[4] |
Higgins JA, Mullinax M, Trussell J, et al. (2011) Sexual satisfaction and sexual health among university students in the United States. Am J Public Health 101: 1643–1654. doi: 10.2105/AJPH.2011.300154
![]() |
[5] | Alizadeh S, Ebadi A, Kariman N, et al. (2018) Dyadic sexual communication scale: psychometrics properties and translation of the Persian version. Sex Relation Ther, 1–12. |
[6] | WHO (2004) Sexual health-a new focus for WHO. Progress in Reproductive Health Research 67: 1–8. |
[7] |
Addis IB, Van Den Eeden SK, Wassel-Fyr CL, et al. (2006) Sexual activity and function in middle-aged and older women. Obstet Gynecol 107: 755–764. doi: 10.1097/01.AOG.0000202398.27428.e2
![]() |
[8] |
Johnson CE (2011) Sexual Health during pregnancy and the postpartum (CME). J Sex Med 8: 1267–1284. doi: 10.1111/j.1743-6109.2011.02223.x
![]() |
[9] | Navidian A, Navabi Rigi S, Imani M, et al. (2016) The effect of sex education on the marital relationship quality of pregnant women. J Hayat 22: 115–127. |
[10] | Bayrami R, Sattarzadeh N, Ranjbar Koocheksarai F , et al. (2009) Eevaluation of sexual behaviors and some of its related factors in pregnant women, Tabriz, IRAN 2005. Urmia Med J 20: 1–7. |
[11] |
Galazka I, Drosdzol-Cop A, Naworska B, et al. (2015) Changes in the sexual function during pregnancy. J Sex Med 12: 445–454. doi: 10.1111/jsm.12747
![]() |
[12] |
Pauls RN, Occhino JA, Dryfhout V, et al. (2008) Effects of pregnancy on pelvic floor dysfunction and body image; a prospective study. Int Urogynecol J 19: 1495–1501. doi: 10.1007/s00192-008-0670-3
![]() |
[13] |
Pauleta JR, Pereira NM, Graca LM (2010) Sexuality during pregnancy. J Sex Med 7: 136–142. doi: 10.1111/j.1743-6109.2009.01538.x
![]() |
[14] |
Vannier SA, Rosen NO (2017) Sexual distress and sexual problems during pregnancy: associations with sexual and relationship satisfaction. J Sex Med 14: 387–395. doi: 10.1016/j.jsxm.2016.12.239
![]() |
[15] | Ozgoli G, Dolatian M , Ozgoli M, et al. (2008) Alteration in sexual drive during pregnancy in women referring to hospitals affilated to Shaheed Beheshti Medical University. Adv Nurs Midwifery 18: 5–12. |
[16] | Pasha H, HadjAhmadi M (2007) Evaluation of sexual behaviors in pregnant women and some related factors. Hormozgan Med J 10: 343–348. |
[17] |
Yu T, Pettit GS, Lansford JE, et al. (2010) The interactive effects of marital conflict and divorce on parent–adult children's relationships. J Marriage Fam 72: 282–292. doi: 10.1111/j.1741-3737.2010.00699.x
![]() |
[18] | Merghati Khoie E, Afshar M, Yavari Kia P, et al. (2012) Sexual belief and behavior of pregnant women referring to public health centers in Karaj-2011. Iran J Obstet Gynecol Infertil 15: 7–14. |
[19] | Nematollahzade M, Maasoumi R, Lamyian M, et al. (2010) Study of women's attitude and sexual function during pregnancy. J Ardabil Univ Med Sci 10: 241–249. |
[20] | Ozgoli G, Khoshabi K, Velaii N, et al. (2006) Knowledge and attitude of pregnant women toward sex during pregnancy and its related factors in general hospitals referring to Shahid Beheshti University of Medical Sciences 2004. J Fam Res 2: 137–147. |
[21] | Heydari M, Kiani Asiabar A, Faghih Zade S (2006) Couples' knowledge and attitude about sexuality in pregnancy. Tehran Univ Med J TUMS Publ 64: 83–89. |
[22] | Abasalizadeh F, Abasalizadeh S (2011) Behavioral dichotomy in sexuality during pregnancy and effect of birth-week intercourse on pregnancy outcomes in an Iranian population. Internet J. Gynecol Obstet 14: 1–6. |
[23] |
Abouzari-Gazafroodi K, Najafi F, Kazemnejad E, et al. (2015) Demographic and obstetric factors affecting women's sexual functioning during pregnancy. Reprod Health 12: 12–72. doi: 10.1186/1742-4755-12-12
![]() |
[24] | Abouzari-Gazafroodi K, Najafi F, Kazemnejad E, et al. (2013) Comparison of sexual function between nulliparous with multiparous pregnant women. J Hayat 18: 55–63. |
[25] | Dadgar S, Karimi FZ, Bakhshi M, et al. (2018) Assessment of sexual dysfunction and its related factors in pregnant women referred to Mashhad health centers (2017–2018). Iran J Obstet Gynecol Infertil 21: 22–29. |
[26] | Hajnasiri H, Aslanbeygi N, Moafi F, et al. (2018) Investigating the relationship between sexual function and mental health in pregnant females. J Nurs Edu 6: 33–40. |
[27] |
Mousazadeh T, Motavalli R (2018) Sexual function and behavior in pregnant women of Ardabil in 2016. J Health Care 20: 40–47. doi: 10.29252/jhc.20.1.40
![]() |
[28] | Tabande A, Behnampour N, Joudi Mashahd M, et al. (2016) Sexual satisfaction of women with gestational diabetes. J Mazandaran Univ Med Sci 26: 202–205. |
[29] | Balali Dehkordi N, Sadat Rouholamini M (2016) The role of body image and obsessive believes in prediction of sexual function among pregnant women. Iran J Obstet Gynecol Infertil 19: 7–16. |
[30] | Jamali S, Mosalanejad L (2013) Sexual dysfnction in Iranian pregnant women. Iran J Reprod Med 11: 479–486. |
[31] | Nik-Azin A, Nainian MR, Zamani M, et al. (2013) Evaluation of sexual function, quality of life, and mental and physical health in pregnant women. J Fam Reprod Health 7: 171–176. |
[32] | Jamali S, Rasekh Jahromi A, Zarei F, et al. (2014) Compression of sexual dysfunction during three trimester of pregnancy in pregnant women who had referred to Peymanieh clinic Jahrom in 2013. Nurs Dev Health 5: 37–45. |
[33] | Bostani Khalesi Z, Rahebi SM, Mansour Ghanaee M (2012) Evaluation of women's sexual performance during first pregnancy. Iran J Obstet Gynecol Infertil 15: 14–20. |
[34] | Torkestani F, Hadavand SH, Khodashenase Z, et al. (2012) Frequency and perception of sexual activity during pregnancy in Iranian couples. Int J Fertil Steril 6: 107–110. |
[35] | Ebrahimian A, Heydari M, Zafarghandi S (2010) Comparison of female sexual dysfunctions before and during pregnancy. Iran J Obstet Gynecol Infertil 13: 30–36. |
[36] | Torkestani F, Hadavand S, Davati A, et al. (2009) Effect of coitus during the second half of pregnancy on pregnancy outcome. Daneshvar Med 16: 5–12. |
[37] | Ozgoli G, Zaki F, Amir Ali Akbari S, et al. (2008) A Survey upon the sexual function and behaviour of pregnant women referring to state health centers of Ahvaz City-2007. Pajoohandeh J 13: 397–403. |
[38] | Bayrami R, Sattarzadeh N, Ranjbar Koochaksariie F, et al. (2008) Sexual dysfunction in couples and its related factors during pregnancy. J Reprod Infertil 9: 271–283. |
[39] | Heidari M, Mohamadi KH, Faghihzade S (2006) Study of changes in sexual activity during pregnancy. Daneshvar Med 13: 27–32. |
[40] | Rahimi S, Seyyed Rasooli E (2004) Sexual behavior during pregnancy: A descriptive study of pregnant women in Tabriz, Iran. Payesh 3: 291–299. |
[41] | Memarian Z, Lamiyan M, Azin A (2016) Levels of sexual satisfaction in third trimester of pregnancy in nulliparous women and related factors. J Mazandaran Univ Med Sci 25: 178–182. |
[42] | Ahmadi Z, Molaie Yarandi E, Malekzadegan A, et al. (2011) Sexual satisfaction and its related factors in primigravidas. Iran J Nur 24: 54–62. |
[43] | Nezal AJ, Samiee RF, Kalhor M, et al. (2018) Sexual quality of life in pregnant women: A cross sectional study. Payesh 17: 421–429. |
[44] | Parhizkar A (2017) Study of the relationship between domestic violence and pregnancy outcomes in mothers referring to Sanandaj comprehensive health centers in 2015–2016. S J Nurs Midwifery Param Fac 2: 33–44. |
[45] |
Sarayloo K, Mirzaei Najmabadi K, Ranjbar F, et al. (2017) Prevalence and risk factors for domestic violence against pregnant women. Iran J Nur 29: 28–35. doi: 10.29252/ijn.29.104.28
![]() |
[46] | Hesami K, Dolatian M, Shams J, et al. (2010) Domestic violence before and during pregnancy among pregnant women. Iran J Nur 23: 51–59. |
[47] | Jahanfar S, Jamshidi R (2002) The prevalence of domestic violence among pregnant women who were attended in Iran university of medical sciences' hospitals. Iran J Nur 15: 93–99. |
[48] |
Abadi MNL, Ghazinour M, Nojomi M, et al. (2012) The buffering effect of social support between domestic violence and self-esteem in pregnant women in Tehran, Iran. J Fam Viol 27: 225–231. doi: 10.1007/s10896-012-9420-x
![]() |
[49] | Taghizadeh Z, Purbakhtyar M, Daneshparvar H, et al. (2015) Comparison the frequency of domestic violence and problem-solving skill among pregnant women with and without violence in Tehran. Iran J Forensic Med 21: 91–98. |
[50] |
Jahanfar S, Malekzadegan Z (2007) The prevalence of domestic violence among pregnant women who were attended in Iran University of Medical Science Hospitals. J Fam Viol 22: 643. doi: 10.1007/s10896-007-9084-0
![]() |
[51] |
Mohammad-Alizadeh-Charandabi S, Bahrami-Vazir E, Kamalifard M, et al. (2016) Intimate partner violence during the first pregnancy: A comparison between adolescents and adults in an urban area of Iran. J Forensic Leg Med 43: 53–60. doi: 10.1016/j.jflm.2016.07.002
![]() |
[52] |
Farrokh-Eslamlou H, Oshnouei S, Haghighi N (2014) Intimate partner violence during pregnancy in Urmia, Iran in 2012. J Forensic Leg Med 24: 28–32. doi: 10.1016/j.jflm.2014.03.007
![]() |
[53] | Noori A, Sanago A, Jouybari L, et al. (2017) Survey of types of domestic violence and its related factors in pregnant mothers in Kalaleh at 2014. Iran J Obstet Gynecol Infertil 19: 54–62. |
[54] | Behnam H, Moghadam Hoseini V, Soltanifar A (2008) Domestic violence against the Iranian pregnant women. Horizon Med Sci 14: 70–76. |
[55] | Moeini B, Ezzati Rastegar K, Hamidi Y, et al. (2018) Social determinants of intimate partner violence among Iranian pregnant women. Koomesh 20: 350–357. |
[56] |
Mohammadhosseini E, Sahraean L, Bahrami T (2010) Domestic abuse before, during and after pregnancy in Jahrom, Islamic Republic of Iran. East Mediterr Health J 16: 752–758. doi: 10.26719/2010.16.7.752
![]() |
[57] | Golchin NAH, Hamzehgardeshi Z, Hamzehgardeshi L, et al. (2014) Sociodemographic characteristics of pregnant women exposed to domestic violence during pregnancy in an Iranian setting. Iran Red Crescent Med J 16. |
[58] | Baheri B, Ziaie M, Mohammadi SZ (2012) Frequency of domestic violence in women with adverse pregnancy outcomes (Karaj 2007–2008). Sci J Hamadan Nurs Midwifery Fac 20: 31–41. |
[59] | Zadeh H, Nouhjah S, Hassan M (2011) Prevalence of domestic violence against pregnant women and its related factors in women referred to health centers in 2010 Ahvaz, Iran. Jentashapir J Health Res 2: 1–9. |
[60] | Ali Kamali M, Rahimi Kian F, Mir Mohamad Ali M, et al. (2015) Comparison of domestic violence and its related factors in pregnant women in both urban and rural population in Zarand city, 2014. J Clin Nurs Midwifery 4: 69–78. |
[61] | Khadivzadeh T, Erfanian F (2011) Comparison of domestic violence during pregnancy with the Pre-pregnancy period and its relating factors. Iran J Obstet Gynecol Infertil 14: 47–56. |
[62] |
Nejatizade AA, Roozbeh N, Yabandeh AP, et al. (2017) Prevalence of domestic violence on pregnant women and maternal and neonatal outcomes in Bandar Abbas, Iran. Electron Physician 9: 5166–5171. doi: 10.19082/5166
![]() |
[63] | Faramarzi M, Esmaelzadeh S, Mosavi S (2005) Prevalence, maternal complications and birth outcome of physical, sexual and emotional domestic violence during pregnancy. Acta Med Iran 43: 115–122. |
[64] | Bagherzadeh R, Keshavarz T, Sharif F, et al. (2008) Relationship between domestic violence during pregnancy and complications of pregnancy, type of delivery and birth weight on delivered women in hospital affiliated to Shiraz University of Medical Sciences. Horizon Med Sci 13: 51–58. |
[65] | Baheri B, Ziaie M, Mohammadi Z (2012) Effect of domestic violence on pregnancy outcomes among pregnant women referring to Karaj Medical Centers. Hakim Health Sys Res 15: 140–146. |
[66] |
Hossieni VM, Toohill J, Akaberi A, et al. (2017) Influence of intimate partner violence during pregnancy on fear of childbirth. Sex Reprod Healthc 14: 17–23. doi: 10.1016/j.srhc.2017.09.001
![]() |
[67] |
Moghaddam Hossieni V, Toohill J, Akaberi A, et al. (2017) Influence of intimate partner violence during pregnancy on fear of childbirth. Sex Reprod Healthc 14: 17–23. doi: 10.1016/j.srhc.2017.09.001
![]() |
[68] | Pazandeh F, Sheikhan Z, Keshavarz Z, et al. (2017) Effects of sex hormones in combined oral contraceptives and cyclofem on female sexual dysfunction score: A study on Iranian Females. Adv Nurs Midwifery 27: 9–14. |
[69] |
Gharacheh M, Azadi S, Mohammadi N, et al. (2016) Domestic violence during pregnancy and women's health-related quality of life. Glob J Health Sci 8: 27–34. doi: 10.5539/gjhs.v8n12p27
![]() |
[70] | Ramezani S, Keramat A, Motaghi Z, et al. (2015) The relationship of sexual satisfaction and marital satisfaction with domestic violence against pregnant women. Int J Pediatr 3: 951–958. |
[71] |
Hassan M, Kashanian M, Roohi M, et al. (2014) Maternal outcomes of intimate partner violence during pregnancy: Study in Iran. Public Health 128: 410–415. doi: 10.1016/j.puhe.2013.11.007
![]() |
[72] | Hassan M, Kashanian M, Hassan M, et al. (2013) Assessment of association between domestic violence during pregnancy with fetal outcome. Iran J Obstet Gynecol Infertil 16: 21–29. |
[73] | Abdollahi F, Yazdani-Cherati J, Majidi Z (2015) Intimate partner violence during pregnancy in the Northern Iran (2010). J Gorgan Univ Med Sci 17: 89–96. |
[74] | Ebrahimi E, Karimian Z, Bonab SKM, et al. (2017) The prevalence of domestic violence and its association with gestational hypertension in pregnant women. Int J Health Stud 3: 21–24. |
[75] | Shakerinezhad M (2013) Domestic violence and related factors in pregnant women. ZUMS J 21: 117–126. |
[76] | Salehi S, Mehralian H (2006) The prevalence and types of domestic violence against pregnant women referred to maternity clinics in Shahrekord, 2003. J Shahrekord Univ Med Sci 8: 72–77. |
[77] | Breuner CC, Mattson G (2016) Sexuality education for children and adolescents. Pediatrics 138. |
[78] |
Anzaku SA, Ogbe EA, Ogbu GI, et al. (2016) Evaluation of changes in sexual response and factors influencing sexuality during pregnancy among Nigerian women in Jos, Nigeria. Int J Reprod Contracept Obstet Gynecol 5: 3576–3582. doi: 10.18203/2320-1770.ijrcog20163448
![]() |
[79] | Malarewicz A, Szymkiewicz J, Rogala J (2006) Sexuality of pregnant women. Ginekol Pol 77: 733–739. |
[80] | Riazi H, BanooZadeh S, MoghimBeigi A, et al. (2013) The effect of sexual health education on sexual function during pregnancy. Payesh 12: 367–374. |
[81] |
Fok WY, Chan LY-S, Yuen PM (2005) Sexual behavior and activity in Chinese pregnant women. Acta Obstet Gynecol Scand 84: 934–938. doi: 10.1111/j.0001-6349.2005.00743.x
![]() |
[82] | Senkumwong N, Chaovisitsaree S, Rugpao S, et al. (2006) The changes of sexuality in Thai women during pregnancy. J Med Assoc Thai 89: 124–129. |
[83] | Merghati Khoie E, Abolghasemi N, Taghdisi MH (2013) Child sexual health: qualitative study, explaining the views of parents. J Sch Public Health Inst Public Health Res 11: 65–74. |
[84] |
Leite AP, Campos AA, Dias AR, et al. (2009) Prevalence of sexual dysfunction during pregnancy. Rev Assoc Med Bras 55: 563–568. doi: 10.1590/S0104-42302009000500020
![]() |
[85] |
Erol B, Sanli O, Korkmaz D, et al. (2007) A cross-sectional study of female sexual function and dysfunction during pregnancy. J Sex Med 4: 1381–1387. doi: 10.1111/j.1743-6109.2007.00559.x
![]() |
[86] |
Ahmed MR, Madny EH, Ahmed WAS (2014) Prevalence of female sexual dysfunction during pregnancy among egyptian women. J Obstet Gynaecol Res 40: 1023–1029. doi: 10.1111/jog.12313
![]() |
[87] | Kuljarusnont S, Russameecharoen K, Thitadilok W (2011) Prevalence of sexual dysfunction in Thai pregnant women. Thai J Obstet Gynaecol 19: 172–180. |
[88] | Stright B (2004) Maternal Neonatal Nursing. Philadelphia Lippincott Williams and Wilkins. |
[89] |
Aslan G, Aslan D, Kizilyar A, et al. (2005) A prospective analysis of sexual functions during pregnancy. Int J Impot Res 17: 154–157. doi: 10.1038/sj.ijir.3901288
![]() |
[90] |
Jamali S, Rasekh JA, Fatmeh Z (2012) Compression domains of sexual response between three trimester of pregnancy. Int J Gynaecol Obstet 119: S594. doi: 10.1016/S0020-7292(12)61384-8
![]() |
[91] |
Schomerus G, Appel K, Meffert PJ, et al. (2013) Personality-related factors as predictors of help-seeking for depression: a population-based study applying the behavioral model of health services use. Soc Psychiatry Psychiatr Epidemiol 48: 1809–1817. doi: 10.1007/s00127-012-0643-1
![]() |
[92] |
Khajehpour M, Simbar M, Jannesari S, et al. (2013) Health status of women with intended and unintended pregnancies. J Public health 127: 58–64. doi: 10.1016/j.puhe.2012.08.011
![]() |
[93] | Gessessew A (2009) Unwanted pregnancy and it's impact on maternal health and utilization of health services in Tigray Region (Adigrat Hospital). Ethiop Med J 47: 1–8. |
[94] |
Serati M, Salvatore S, Siesto G, et al. (2010) Female sexual function during pregnancy and after childbirth. J Sex Med 7: 2782–2790. doi: 10.1111/j.1743-6109.2010.01893.x
![]() |
[95] |
Brotto L, Atallah S, Johnson-Agbakwu C, et al. (2016) Psychological and interpersonal dimensions of sexual function and dysfunction. J Sex Med 13: 538–571. doi: 10.1016/j.jsxm.2016.01.019
![]() |
[96] |
Van Minnen A, Kampman MJS, Therapy R (2000) The interaction between anxiety and sexual functioning: A controlled study of sexual functioning in women with anxiety disorders. Sex Relation Ther 15: 47–57. doi: 10.1080/14681990050001556
![]() |
[97] |
Heider N, Spruyt A, De Houwer J (2015) Implicit beliefs about ideal body image predict body image dissatisfaction. Front Psychol 6: 1402. doi: 10.3389/fpsyg.2015.01402
![]() |
[98] |
Mehta UJ, Siega-Riz AM, Herring AH (2011) Effect of body image on pregnancy weight gain. Matern Child Health J 15: 324–332. doi: 10.1007/s10995-010-0578-7
![]() |
[99] | Solimany A, Delpisheh A, Khademi N, et al. (2016) Prevalence of violence against women in during pregnancy in IRAN: A systematic review and meta-analysis. J Urmia Nurs Midwifery Fac 13: 973–986. |
[100] | Shamu S, Abrahams N, Temmerman M, et al. (2011) A systematic review of African Studies on intimate partner violence against pregnant women: Prevalence and risk factors. PLoS One 6. |
[101] |
Ergönen AT, Özdemir MH, Can İÖ, et al. (2009) Domestic violence on pregnant women in Turkey. J Forensic Leg Med 16: 125–129. doi: 10.1016/j.jflm.2008.08.009
![]() |
[102] |
Hammoury N, Khawaja M (2007) Screening for domestic violence during pregnancy in an antenatal clinic in Lebanon. Eur J Public Health 17: 605–606. doi: 10.1093/eurpub/ckm009
![]() |
[103] | Afifi ZE, Al-Muhaideb NS, Hadish NF, et al. (2011) Domestic violence and its impact on married women's health in Eastern Saudi Arabia. Saudi Med J 32: 612–620. |
[104] | Soleimani M, Jamshidimanesh M, Daneshkojuri M, et al. (2012) Correlation between partner violence and preterm labor. J Qazvin Univ Med Sci 15: 53–59. |
[105] | Cavalin C (2010) WHO Multi-country study on women's health and domestic violence against women. Initial results on prevalence, health outcomes and women's responses. JSTOR, 837–839. Available form: https://www.who.int/reproductivehealth/publications/violence/24159358X/en/. |
[106] |
Silverman JG, Decker MR, Reed E, et al. (2006) Intimate partner violence victimization prior to and during pregnancy among women residing in 26 US states: Associations with maternal and neonatal health. Am J Obstet Gynecol 195: 140–148. doi: 10.1016/j.ajog.2005.12.052
![]() |
[107] |
Records K (2007) A critical review of maternal abuse and infant outcomes: Implications for newborn nurses. Newborn Infant Nurs Rev 7: 7–13. doi: 10.1053/j.nainr.2006.12.005
![]() |
[108] |
Hill A, Pallitto C, McCleary‐Sills J, et al. (2016) A systematic review and meta‐analysis of intimate partner violence during pregnancy and selected birth outcomes. Int J Gynaecol Obstet 133: 269–276. doi: 10.1016/j.ijgo.2015.10.023
![]() |
[109] |
Han A, Stewart DE (2014) Maternal and fetal outcomes of intimate partner violence associated with pregnancy in the Latin American and Caribbean region. Int J Gynaecol Obstet 124: 6–11. doi: 10.1016/j.ijgo.2013.06.037
![]() |
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Sample size | β | $ E[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ E[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ |
n = 10 | 1 | 0.633799 | 0.0618118 | 0.636254 | 0.086091 |
2 | 0.471249 | 0.0302985 | 0.57848 | 0.0651343 | |
3 | 0.368875 | 0.0174016 | 0.530634 | 0.056318 | |
n = 20 | 1 | 0.675683 | 0.0343049 | 0.027132 | 0.617761 |
2 | 0.493352 | 0.0160719 | 0.55787 | 0.0203291 | |
3 | 0.38382 | 0.00911087 | 0.507678 | 0.017622 | |
n = 30 | 1 | 0.689803 | 0.023654 | 0.611674 | 0.0135907 |
2 | 0.500628 | 0.0109196 | 0.550958 | 0.0101643 | |
3 | 0.388709 | 0.00616504 | 0.499906 | 0.0061073 | |
n = 40 | 1 | 0.696893 | 0.0180386 | 0.608645 | 0.00826977 |
2 | 0.504248 | 0.0082666 | 0.547493 | 0.00618269 | |
3 | 0.391136 | 0.00465795 | 0.495995 | 0.00438061 | |
n = 50 | 1 | 0.701156 | 0.014575 | 0.606833 | 0.00560691 |
2 | 0.506414 | 0.00665019 | 0.545411 | 0.00419222 | |
3 | 0.392587 | 0.00374276 | 0.493641 | 0.00365316 |
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
Normal | [0.74, 1.2] | [74,112] | [269,418] | [81,267] | [29,273] |
Chemical diabetic | [0.83, 1.2] | [75,114] | [413,643] | [109,748] | [60,300] |
Overt diabetic | [0.74, 1.2] | [120,353] | [538, 1520] | [10,460] | [150,458] |
(II) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \mathbb{P}(\text{normal}) $ | 0.319397 | 0.45749 | 0.318917 | 0.395996 | 0.329123 |
$ \mathbb{P}(\text{chemical diabetic}) $ | 0.361206 | 0.483345 | 0.603506 | 0.234745 | 0.405461 |
$ \mathbb{P}(\text{overt diabetic}) $ | 0.319397 | 0.0591652 | 0.0775768 | 0.369259 | 0.265417 |
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \beta=1 $ | 0.945856 | 0.748902 | 0.722495 | 0.922879 | 0.931814 |
$ \beta=2 $ | 0.71489 | 0.604572 | 0.5662 | 0.699949 | 0.705139 |
$ \beta=3 $ | 0.539139 | 0.488135 | 0.4556 | 0.531995 | 0.534171 |
(II) Item | $ W^{*}(\Lambda_{1}) $ | $ W^{*}(\Lambda_{2}) $ | $ W^{*}(\Lambda_{3}) $ | $ W^{*}(\Lambda_{4}) $ | $ W^{*}(\Lambda_{5}) $ |
$ \beta=1 $ | 0.181642 | 0.221183 | 0.227102 | 0.185864 | 0.18421 |
$ \beta=2 $ | 0.188615 | 0.210614 | 0.218853 | 0.191454 | 0.190463 |
$ \beta=3 $ | 0.194114 | 0.204272 | 0.211027 | 0.195506 | 0.195081 |
Method | Normal | Chemical diabetic | Overt diabetic | In general |
Entropy method | 100% | 55.5% | 60.6% | 72.03% |
Generalized exponential entropy | ||||
method | 100% | 44.4% | 33.3% | 59.23% |
Sample size | β | $ E[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}(\widehat{F}_{n})] $ | $ E[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ | $ Var[RGEXn_{\beta}^{*}(\widehat{F}_{n})] $ |
n = 10 | 1 | 0.633799 | 0.0618118 | 0.636254 | 0.086091 |
2 | 0.471249 | 0.0302985 | 0.57848 | 0.0651343 | |
3 | 0.368875 | 0.0174016 | 0.530634 | 0.056318 | |
n = 20 | 1 | 0.675683 | 0.0343049 | 0.027132 | 0.617761 |
2 | 0.493352 | 0.0160719 | 0.55787 | 0.0203291 | |
3 | 0.38382 | 0.00911087 | 0.507678 | 0.017622 | |
n = 30 | 1 | 0.689803 | 0.023654 | 0.611674 | 0.0135907 |
2 | 0.500628 | 0.0109196 | 0.550958 | 0.0101643 | |
3 | 0.388709 | 0.00616504 | 0.499906 | 0.0061073 | |
n = 40 | 1 | 0.696893 | 0.0180386 | 0.608645 | 0.00826977 |
2 | 0.504248 | 0.0082666 | 0.547493 | 0.00618269 | |
3 | 0.391136 | 0.00465795 | 0.495995 | 0.00438061 | |
n = 50 | 1 | 0.701156 | 0.014575 | 0.606833 | 0.00560691 |
2 | 0.506414 | 0.00665019 | 0.545411 | 0.00419222 | |
3 | 0.392587 | 0.00374276 | 0.493641 | 0.00365316 |
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
Normal | [0.74, 1.2] | [74,112] | [269,418] | [81,267] | [29,273] |
Chemical diabetic | [0.83, 1.2] | [75,114] | [413,643] | [109,748] | [60,300] |
Overt diabetic | [0.74, 1.2] | [120,353] | [538, 1520] | [10,460] | [150,458] |
(II) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \mathbb{P}(\text{normal}) $ | 0.319397 | 0.45749 | 0.318917 | 0.395996 | 0.329123 |
$ \mathbb{P}(\text{chemical diabetic}) $ | 0.361206 | 0.483345 | 0.603506 | 0.234745 | 0.405461 |
$ \mathbb{P}(\text{overt diabetic}) $ | 0.319397 | 0.0591652 | 0.0775768 | 0.369259 | 0.265417 |
(I) Item | $ \Lambda_{1} $ | $ \Lambda_{2} $ | $ \Lambda_{3} $ | $ \Lambda_{4} $ | $ \Lambda_{5} $ |
$ \beta=1 $ | 0.945856 | 0.748902 | 0.722495 | 0.922879 | 0.931814 |
$ \beta=2 $ | 0.71489 | 0.604572 | 0.5662 | 0.699949 | 0.705139 |
$ \beta=3 $ | 0.539139 | 0.488135 | 0.4556 | 0.531995 | 0.534171 |
(II) Item | $ W^{*}(\Lambda_{1}) $ | $ W^{*}(\Lambda_{2}) $ | $ W^{*}(\Lambda_{3}) $ | $ W^{*}(\Lambda_{4}) $ | $ W^{*}(\Lambda_{5}) $ |
$ \beta=1 $ | 0.181642 | 0.221183 | 0.227102 | 0.185864 | 0.18421 |
$ \beta=2 $ | 0.188615 | 0.210614 | 0.218853 | 0.191454 | 0.190463 |
$ \beta=3 $ | 0.194114 | 0.204272 | 0.211027 | 0.195506 | 0.195081 |
Method | Normal | Chemical diabetic | Overt diabetic | In general |
Entropy method | 100% | 55.5% | 60.6% | 72.03% |
Generalized exponential entropy | ||||
method | 100% | 44.4% | 33.3% | 59.23% |